Glomerular aging in females is a multi-stage reversible process mediated by phenotypic changes in progenitors. (Q35085046)

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4 January 2020

title

Glomerular aging in females is a multi-stage reversible process mediated by phenotypic changes in progenitors (English)

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4 January 2020

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6

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Mylene Potier

7

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4 January 2020

Andrew Koff

9

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4 January 2020

author name string

Zheng Feng

1

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4 January 2020

Anna Rita Plati

2

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4 January 2020

Qing-li Cheng

3

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Mariana Berho

4

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Anita Banerjee

5

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Wen-che Jy

8

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Liliane J Striker

10

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Gary E Striker

11

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4 January 2020

language of work or name

English

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publication date

1 August 2005

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The American Journal of Pathology

4 January 2020

published in

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volume

167

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issue

2

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4 January 2020

page(s)

355-363

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Rejuvenation of aged progenitor cells by exposure to a young systemic environment

4 January 2020

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Resistance to glomerulosclerosis in B6 mice disappears after menopause

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Upregulation of type I collagen by TGF-beta in mesangial cells is blocked by PPARgamma activation

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Glomerulosclerosis is transmitted by bone marrow-derived mesangial cell progenitors

8 July 2018

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Bone marrow is a reservoir of repopulating mesangial cells during glomerular remodeling.

8 July 2018

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High glucose-induced hypertrophy of mesangial cells requires p27(Kip1), an inhibitor of cyclin-dependent kinases

8 July 2018

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8 July 2018

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Dissociation of glomerular hypertrophy, cell proliferation, and glomerulosclerosis in mouse strains heterozygous for a mutation (Os) which induces a 50% reduction in nephron number

8 July 2018

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Relationships between mesangial cell proliferation and types I and IV collagen mRNA levels in vitro

8 July 2018

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Progressive glomerulosclerosis develops in transgenic mice chronically expressing growth hormone and growth hormone releasing factor but not in those expressing insulinlike growth factor-1

8 July 2018

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Development of albuminuria and glomerular lesions in normoglycemic B6 recipients of db/db mice bone marrow: the role of mesangial cell progenitors.

8 July 2018

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The lack of cyclin kinase inhibitor p27(Kip1) ameliorates progression of diabetic nephropathy

27 September 2018

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The cyclin kinase inhibitor p21WAF1, CIP1 is increased in experimental diabetic nephropathy: potential role in glomerular hypertrophy.

27 September 2018

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Crossref

https://api.crossref.org/works/10.1016%2FS0002-9440%2810%2962981-1

Oxidant/Antioxidant Balance in Isolated Glomeruli and Cultured Mesangial Cells

7 January 2021

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HGF-induced tubulogenesis and branching of epithelial cells is modulated by extracellular matrix and TGF-beta

7 January 2021

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7 January 2021

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PubMed

https://pubmed.ncbi.nlm.nih.gov/16049323

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Mesangial cells from transgenic mice with progressive glomerulosclerosis exhibit stable, phenotypic changes including undetectable MMP-9 and increased type IV collagen

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/16049323

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Glomerular lesions in nonobese diabetic mouse: before and after the onset of hyperglycemia

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/16049323

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1016/S0002-9440(10)62981-1

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:16049323%20AND%20SRC:MED&resulttype=core&format=json

4 January 2020

1 reference

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4 January 2020

PubMed publication ID

16049323

1 reference