Pancreatic trypsin increases matrix metalloproteinase-9 accumulation and activation during acute intestinal ischemia-reperfusion in the rat. (Q35098124)

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English	Pancreatic trypsin increases matrix metalloproteinase-9 accumulation and activation during acute intestinal ischemia-reperfusion in the rat.	scientific article

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scholarly article

1 reference

Europe PubMed Central

6 August 2017

Pancreatic trypsin increases matrix metalloproteinase-9 accumulation and activation during acute intestinal ischemia-reperfusion in the rat. (English)

1 reference

Europe PubMed Central

6 August 2017

author name string

Henrique S Rosário

1

1 reference

Europe PubMed Central

6 August 2017

Stephen W Waldo

2

1 reference

Europe PubMed Central

6 August 2017

Scott A Becker

3

1 reference

Europe PubMed Central

6 August 2017

Geert W Schmid-Schönbein

4

1 reference

Europe PubMed Central

6 August 2017

language of work or name

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publication date

1 May 2004

1 reference

Europe PubMed Central

6 August 2017

The American Journal of Pathology

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Europe PubMed Central

6 August 2017

164

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Europe PubMed Central

6 August 2017

5

1 reference

Europe PubMed Central

6 August 2017

1707-1716

1 reference

Europe PubMed Central

6 August 2017

Pathogenesis of multiple organ dysfunction syndrome--endotoxin, inflammatory cells, and their mediators: cytokines and reactive oxygen species

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Gelatinase B deficiency protects against endotoxin shock

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The gastrointestinal tract in critical illness

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Gelatinase B: a tuner and amplifier of immune functions.

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9 July 2018

Gelatinase B functions as regulator and effector in leukocyte biology

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Pancreatic enzymes and microvascular cell activation in multiorgan failure

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Estimation of gelatinase content in rat brain: effect of focal ischemia

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The endothelin system in septic and endotoxin shock

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Neutrophil gelatinase B potentiates interleukin-8 tenfold by aminoterminal processing, whereas it degrades CTAP-III, PF-4, and GRO-alpha and leaves RANTES and MCP-2 intact.

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The pancreas as a source of cardiovascular cell activating factors

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Regulation of tissue injury responses by the exposure of matricryptic sites within extracellular matrix molecules

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9 July 2018

Generation of in vivo activating factors in the ischemic intestine by pancreatic enzymes

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Proteinase-activated receptor-2-activating peptides induce leukocyte rolling, adhesion, and extravasation in vivo

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9 July 2018

Matrix metalloproteinases

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Activation of type IV procollagenases by human tumor-associated trypsin-2

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Focalized proteolysis: spatial and temporal regulation of extracellular matrix degradation at the cell surface

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Progelatinase B forms from human neutrophils. complex formation of monomer/lipocalin with TIMP-1.

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Dog mastocytoma cells secrete a 92-kD gelatinase activated extracellularly by mast cell chymase

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Shock and multiple organ failure.

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Isolation and primary structure of NGAL, a novel protein associated with human neutrophil gelatinase.

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Two types of human mast cells that have distinct neutral protease compositions

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Role of xanthine oxidase and granulocytes in ischemia-reperfusion injury

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The cysteine switch: a principle of regulation of metalloproteinase activity with potential applicability to the entire matrix metalloproteinase gene family

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Pancreatic enzymes sustain systemic inflammation after an initial endotoxin challenge.

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Identifiers

10.1016/S0002-9440(10)63729-7

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

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