Deficiency of MGAT2 increases energy expenditure without high-fat feeding and protects genetically obese mice from excessive weight gain (Q35152233)

6 August 2017

title

Deficiency of MGAT2 increases energy expenditure without high-fat feeding and protects genetically obese mice from excessive weight gain (English)

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6 August 2017

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David W Nelson

1

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6 August 2017

Yu Gao

2

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6 August 2017

Nicole M Spencer

3

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6 August 2017

Taylor Banh

4

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6 August 2017

Chi-Liang Eric Yen

5

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6 August 2017

publication date

6 July 2011

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Journal of Lipid Research

6 August 2017

published in

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6 August 2017

volume

52

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6 August 2017

issue

9

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6 August 2017

page(s)

1723-1732

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Bowels control brain: gut hormones and obesity

6 August 2017

cites work

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13 July 2018

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Deficiency of the intestinal enzyme acyl CoA:monoacylglycerol acyltransferase-2 protects mice from metabolic disorders induced by high-fat feeding.

13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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Properties of the mouse intestinal acyl-CoA:monoacylglycerol acyltransferase, MGAT2.

13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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13 July 2018

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12 December 2020

Brown Adipose Tissue: Thermic Response Increased by a Single Low Protein, High Carbohydrate Meal

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12 December 2020

Reduced thermic effect of feeding in obesity: role of norepinephrine

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12 December 2020

Diet-induced thermogenesis measured over a whole day in obese and nonobese women

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12 December 2020

Role of MGAT2 and DGAT1 in the release of gut peptides after triglyceride ingestion

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12 December 2020

Differential responses of yellow Avy/A and agouti A/a (BALB/c X VY) F1 hybrid mice to the same diets: glucose tolerance, weight gain, and adipocyte cellularity

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12 December 2020

Identifiers

DOI

10.1194/JLR.M016840

1 reference

6 August 2017

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6 August 2017

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