MPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP. (Q35175367)

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English	MPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP.	scientific article

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9 February 2020

MPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP (English)

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9 February 2020

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9 February 2020

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9 February 2020

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9 February 2020

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Krista K Hill

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9 February 2020

Holly Filak

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9 February 2020

Jennifer Mogan

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9 February 2020

Heather Knowles

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9 February 2020

Bicheng Zhang

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9 February 2020

Anne-Laure Perraud

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9 February 2020

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3 August 2011

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9 February 2020

Journal of Immunology

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9 February 2020

187

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9 February 2020

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9 February 2020

2595-2601

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9 February 2020

Production of IFN-β during Listeria monocytogenes infection is restricted to monocyte/macrophage lineage

1 reference

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A fluorescence reporter model defines "Tip-DCs" as the cellular source of interferon β in murine listeriosis

1 reference

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28 July 2018

Antagonistic crosstalk between type I and II interferons and increased host susceptibility to bacterial infections

1 reference

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28 July 2018

Cellular reactive oxygen species inhibit MPYS induction of IFNβ

1 reference

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The ubiquitin ligase TRIM56 regulates innate immune responses to intracellular double-stranded DNA

1 reference

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IFI16 is an innate immune sensor for intracellular DNA

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28 July 2018

Type I interferon: friend or foe?

1 reference

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Cutting edge: Cytosolic bacterial DNA activates the inflammasome via Aim2.

1 reference

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28 July 2018

c-di-AMP secreted by intracellular Listeria monocytogenes activates a host type I interferon response

1 reference

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1 reference

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Absent in melanoma 2 is required for innate immune recognition of Francisella tularensis.

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Listeria monocytogenes triggers AIM2-mediated pyroptosis upon infrequent bacteriolysis in the macrophage cytosol.

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Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population

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The AIM2 inflammasome is critical for innate immunity to Francisella tularensis

1 reference

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Listeria monocytogenes is sensed by the NLRP3 and AIM2 inflammasome

1 reference

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28 July 2018

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1 reference

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28 July 2018

Induction of IFN-alphabeta enables Listeria monocytogenes to suppress macrophage activation by IFN-gamma

1 reference

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28 July 2018

STING regulates intracellular DNA-mediated, type I interferon-dependent innate immunity

1 reference

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28 July 2018

A host type I interferon response is induced by cytosolic sensing of the bacterial second messenger cyclic-di-GMP

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3159690

28 July 2018

ERIS, an endoplasmic reticulum IFN stimulator, activates innate immune signaling through dimerization

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3159690

28 July 2018

Characterization of the interferon-producing cell in mice infected with Listeria monocytogenes

1 reference

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28 July 2018

STING is an endoplasmic reticulum adaptor that facilitates innate immune signalling

1 reference

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c-di-GMP is an effective immunomodulator and vaccine adjuvant against pneumococcal infection.

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Listeria monocytogenes multidrug resistance transporters activate a cytosolic surveillance pathway of innate immunity

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28 July 2018

MPYS, a novel membrane tetraspanner, is associated with major histocompatibility complex class II and mediates transduction of apoptotic signals

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Type I interferon production enhances susceptibility to Listeria monocytogenes infection

1 reference

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12 December 2020

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Restriction of Legionella pneumophila growth in macrophages requires the concerted action of cytokine and Naip5/Ipaf signalling pathways

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12 December 2020

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9 February 2020

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