Altered protein folding may be the molecular basis of most cases of cystic fibrosis (Q35344873)

7 August 2017

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title

Altered protein folding may be the molecular basis of most cases of cystic fibrosis (English)

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7 August 2017

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Thomas PJ

1

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7 August 2017

Ko YH

series ordinal

2

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7 August 2017

Pedersen PL

series ordinal

3

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7 August 2017

language of work or name

English

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publication date

1 November 1992

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7 August 2017

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7 August 2017

volume

312

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7 August 2017

issue

1

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7 August 2017

page(s)

7-9

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Identification of the cystic fibrosis gene: cloning and characterization of complementary DNA

7 August 2017

cites work

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https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

A cluster of cystic fibrosis mutations in the first nucleotide-binding fold of the cystic fibrosis conductance regulator protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Correction of the cystic fibrosis defect in vitro by retrovirus-mediated gene transfer

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Expression of the cystic fibrosis gene in non-epithelial invertebrate cells produces a regulated anion conductance

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Generation of cAMP-Activated Chloride Currents by Expression of CFTR

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Chloride Conductance Expressed by ΔF508 and Other Mutant CFTRs In Xenopus Oocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

A kinetic study of the competition between renaturation and aggregation during the refolding of denatured-reduced egg white lysozyme.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Targeting of cell-surface beta-amyloid precursor protein to lysosomes: alternative processing into amyloid-bearing fragments.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Production of recombinant human transthyretin with biological activities toward the understanding of the molecular basis of familial amyloidotic polyneuropathy (FAP).

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Amphotericin B treatment dissociates in vivo replication of the scrapie agent from PrP accumulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

The mechanism of Z alpha 1-antitrypsin accumulation in the liver

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Altered chloride ion channel kinetics associated with the ΔF508 cystic fibrosis mutation

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Expression of cystic fibrosis transmembrane conductance regulator corrects defective chloride channel regulation in cystic fibrosis airway epithelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

Cystic Fibrosis Transmembrane Conductance Regulator: Nucleotide Binding to a Synthetic Peptide

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2F0014-5793%2892%2981399-7

10.1016/0014-5793(92)81399-7

21 January 2018

Identifiers

DOI

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7 August 2017

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