Ischemia/reperfusion-induced CHOP expression promotes apoptosis and impairs renal function recovery: the role of acidosis and GPR4 (Q35364307)

8 August 2017

title

Ischemia/reperfusion-induced CHOP expression promotes apoptosis and impairs renal function recovery: the role of acidosis and GPR4 (English)

1 reference

8 August 2017

Biao Dong

1

1 reference

8 August 2017

Honglan Zhou

2

1 reference

8 August 2017

Conghui Han

3

1 reference

8 August 2017

Jufang Yao

4

1 reference

8 August 2017

Longmei Xu

5

1 reference

8 August 2017

Ming Zhang

6

1 reference

8 August 2017

Yaowen Fu

7

1 reference

8 August 2017

Qiang Xia

8

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8 August 2017

language of work or name

English

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publication date

24 October 2014

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8 August 2017

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8 August 2017

volume

9

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8 August 2017

issue

10

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8 August 2017

page(s)

e110944

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Creative Commons Attribution 4.0 International

8 August 2017

copyright license

start time

24 October 2014

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

0 references

cites work

The stress-regulated transcription factor CHOP promotes hepatic inflammatory gene expression, fibrosis, and oncogenesis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Ischemia-reperfusion induces renal tubule pyroptosis via the CHOP-caspase-11 pathway

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Acidosis activation of the proton-sensing GPR4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysis

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

CHOP deficiency results in elevated lipopolysaccharide-induced inflammation and kidney injury

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

C/EBP homologous protein (CHOP) deficiency aggravates hippocampal cell apoptosis and impairs memory performance

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Endoplasmic reticulum stress modulates liver inflammatory immune response in the pathogenesis of liver ischemia and reperfusion injury

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Transient ureteral obstruction prevents against kidney ischemia/reperfusion injury via hypoxia-inducible factor (HIF)-2α activation

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Activation of GPR4 by acidosis increases endothelial cell adhesion through the cAMP/Epac pathway

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

PKR-dependent CHOP induction limits hyperoxia-induced lung injury.

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Deletion of the pH sensor GPR4 decreases renal acid excretion

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

The endoplasmic reticulum stress response factor CHOP-10 protects against hypoxia-induced neuronal death.

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Vascular abnormalities in mice deficient for the G protein-coupled receptor GPR4 that functions as a pH sensor

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Endoplasmic reticulum stress signaling in disease

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Mediators of ischemic renal injury

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

CHOP regulates the p53-MDM2 axis and is required for neuronal survival after seizures

12 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Atypical activation of the unfolded protein response in cystic fibrosis airway cells contributes to p38 MAPK-mediated innate immune responses.

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Spectrum of acute renal failure in the intensive care unit: the PICARD experience

26 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4208823

Ischemia-induced neuronal cell death is mediated by the endoplasmic reticulum stress pathway involving CHOP

26 September 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/25343248

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

C/EBP homologous protein deficiency attenuates myocardial reperfusion injury by inhibiting myocardial apoptosis and inflammation

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/25343248

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Lipopolysaccharide-induced cross-tolerance against renal ischemia-reperfusion injury is mediated by hypoxia-inducible factor-2α-regulated nitric oxide production

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/25343248

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PONE.0110944

1 reference

8 August 2017

0 references

1 reference

8 August 2017

1 reference