Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury (Q35640549)

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scientific article published on 27 December 2011

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English	Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury	scientific article published on 27 December 2011

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19 January 2020

Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury (English)

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reperfusion injury

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Merry L. Lindsey

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Christine E. Seidman

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Christine E Seidman

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Jun Yoshioka

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William A Chutkow

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Samuel Lee

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Jae Bum Kim

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Jie Yan

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Edward P Feener

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Jonathan G Seidman

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Richard T Lee

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27 December 2011

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Journal of Clinical Investigation

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19 January 2020

Pivotal role of cardiomyocyte TGF-β signaling in the murine pathological response to sustained pressure overload

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Diminished AMPK signaling response to fasting in thioredoxin-interacting protein knockout mice

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Deletion of the alpha-arrestin protein Txnip in mice promotes adiposity and adipogenesis while preserving insulin sensitivity

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Intracellular shuttling and mitochondrial function of thioredoxin-interacting protein

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Mitochondrial permeability transition pore opening as a promising therapeutic target in cardiac diseases

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Attenuation of angiotensin II-induced vascular dysfunction and hypertension by overexpression of Thioredoxin 2.

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Endothelial-specific expression of mitochondrial thioredoxin promotes ischemia-mediated arteriogenesis and angiogenesis

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Identification of thioredoxin-2 as a regulator of the mitochondrial permeability transition

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Glucose sensing by MondoA:Mlx complexes: a role for hexokinases and direct regulation of thioredoxin-interacting protein expression

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Role of mitochondrial dysfunction in insulin resistance

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Hypoxic reprogramming

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Myocardial reperfusion injury.

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TXNIP regulates peripheral glucose metabolism in humans

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Role of thioredoxin in cell growth through interactions with signaling molecules

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Modulation of electron transport protects cardiac mitochondria and decreases myocardial injury during ischemia and reperfusion.

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Different mechanisms of mitochondrial proton leak in ischaemia/reperfusion injury and preconditioning: implications for pathology and cardioprotection

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Inhibition of endogenous thioredoxin in the heart increases oxidative stress and cardiac hypertrophy

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Human mitochondrial thioredoxin. Involvement in mitochondrial membrane potential and cell death

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Targeted deletion of thioredoxin-interacting protein regulates cardiac dysfunction in response to pressure overload

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Long-Term Effects of Increased Glucose Entry on Mouse Hearts During Normal Aging and Ischemic Stress

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12 December 2020

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10.1172/JCI44927

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19 January 2020

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19 January 2020

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19 January 2020

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