NMDA and nitric oxide act through the cGMP signal transduction pathway to repress hypothalamic gonadotropin-releasing hormone gene expression (Q35844187)

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scientific article published in February 1996

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English	NMDA and nitric oxide act through the cGMP signal transduction pathway to repress hypothalamic gonadotropin-releasing hormone gene expression	scientific article published in February 1996

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scholarly article

1 reference

Europe PubMed Central

10 August 2017

NMDA and nitric oxide act through the cGMP signal transduction pathway to repress hypothalamic gonadotropin-releasing hormone gene expression (English)

1 reference

Europe PubMed Central

10 August 2017

author name string

D D Belsham

1

1 reference

Europe PubMed Central

10 August 2017

W C Wetsel

2

1 reference

Europe PubMed Central

10 August 2017

P L Mellon

3

1 reference

Europe PubMed Central

10 August 2017

language of work or name

0 references

publication date

1 February 1996

1 reference

Europe PubMed Central

10 August 2017

The EMBO Journal

1 reference

Europe PubMed Central

10 August 2017

15

1 reference

Europe PubMed Central

10 August 2017

3

1 reference

Europe PubMed Central

10 August 2017

538-547

1 reference

Europe PubMed Central

10 August 2017

Protein measurement with the Folin phenol reagent

1 reference

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11 July 2018

Expression of candidate pro-GnRH processing enzymes in rat hypothalamus and an immortalized hypothalamic neuronal cell line.

1 reference

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Regulation of gonadotropin-releasing hormone by protein kinase-A and -C in immortalized hypothalamic neurons

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The cGMP-dependent protein kinase--gene, protein, and function

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Evidence for transcriptional inhibition of GnRH gene expression by phorbol ester at a proximal promoter region

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N-methyl-D-aspartate receptor gene expression in the hamster hypothalamus and in immortalized luteinizing hormone-releasing hormone neurones

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N-methyl-D-aspartate (NMDA) receptors mediate the release of gonadotropin-releasing hormone (GnRH) by NMDA in a hypothalamic GnRH neuronal cell line (GT1-1).

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Nitric oxide regulates luteinizing hormone-releasing hormone secretion

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Nitric oxide: a new messenger in the brain

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Hypothalamus-specific regulation of gonadotropin-releasing hormone gene expression.

1 reference

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Nitric oxide and cGMP analogs activate transcription from AP-1-responsive promoters in mammalian cells

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Calcium signaling in neurons: molecular mechanisms and cellular consequences

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Role of nitric oxide in the control of luteinizing hormone-releasing hormone release in vivo and in vitro

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Structural diversity of neuronal nitric oxide synthase mRNA in the nervous system

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A neuron-specific enhancer targets expression of the gonadotropin-releasing hormone gene to hypothalamic neurosecretory neurons

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The POU homeodomain transcription factor Oct-1 is essential for activity of the gonadotropin-releasing hormone neuron-specific enhancer

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Second messenger regulation of mouse gonadotropin-releasing hormone gene expression in immortalized mouse hypothalamic GT1–3 cells

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Nitric oxide synthesized by gonadotropin-releasing hormone neurons is a mediator of N-methyl-D-aspartate (NMDA)-induced GnRH secretion

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Involvement of nitric oxide in the regulation of gonadotropin-releasing hormone release from the GT1-1 neuronal cell line

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Regulation of gonadotropin-releasing hormone transcription by protein kinase C is mediated by evolutionarily conserved promoter-proximal elements

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1 reference

Europe PubMed Central

10 August 2017

1 reference

Europe PubMed Central

10 August 2017

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