Gamma-aminobutyric acid type A receptors modulate cAMP-mediated long-term potentiation and long-term depression at monosynaptic CA3-CA1 synapses (Q35871597)

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scientific article published on 10 April 2001

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English	Gamma-aminobutyric acid type A receptors modulate cAMP-mediated long-term potentiation and long-term depression at monosynaptic CA3-CA1 synapses	scientific article published on 10 April 2001

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6 November 2019

Gamma-aminobutyric acid type A receptors modulate cAMP-mediated long-term potentiation and long-term depression at monosynaptic CA3-CA1 synapses (English)

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6 November 2019

long-term potentiation

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3

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author name string

T P Yu

1

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S McKinney

2

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N Davidson

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10 April 2001

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Proceedings of the National Academy of Sciences of the United States of America

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98

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5264-5269

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6 November 2019

Capture of a protein synthesis-dependent component of long-term depression

1 reference

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Inhibition of the cAMP pathway decreases early long-term potentiation at CA1 hippocampal synapses.

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Lamina-specific synaptic activation causes domain-specific alterations in dendritic immunostaining for MAP2 and CAM kinase II.

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Long-term depression in the hippocampus in vivo is associated with protein phosphatase-dependent alterations in extracellular signal-regulated kinase

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12 December 2020

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Effects of A1 and A2 adenosine receptor antagonists on the induction and reversal of long-term potentiation in guinea pig hippocampal slices of CA1 neurons

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nMDA receptor activation increases cyclic AMP in area CA1 of the hippocampus via calcium/calmodulin stimulation of adenylyl cyclase

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Physiology and pharmacology of unitary synaptic connections between pairs of cells in areas CA3 and CA1 of rat hippocampal slice cultures

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https://pubmed.ncbi.nlm.nih.gov/11296264

12 December 2020

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Identifiers

10.1073/PNAS.091093998

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6 November 2019

2001PNAS...98.5264Y

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6 November 2019

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