Acetyl-CoA carboxylase 2-/- mutant mice are protected against fatty liver under high-fat, high-carbohydrate dietary and de novo lipogenic conditions. (Q35874567)

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scientific article published on 23 February 2012

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English	Acetyl-CoA carboxylase 2-/- mutant mice are protected against fatty liver under high-fat, high-carbohydrate dietary and de novo lipogenic conditions.	scientific article published on 23 February 2012

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24 January 2020

Acetyl-CoA carboxylase 2-/- mutant mice are protected against fatty liver under high-fat, high-carbohydrate dietary and de novo lipogenic conditions (English)

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24 January 2020

Lutfi Abu-Elheiga

1

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24 January 2020

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Hongmei Wu

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24 January 2020

Ziwei Gu

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24 January 2020

Rubin Bressler

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24 January 2020

Salih J Wakil

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23 February 2012

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24 January 2020

Journal of Biological Chemistry

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24 January 2020

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12578-12588

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Gene knockout of Acc2 has little effect on body weight, fat mass, or food intake

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Recent advances in nonalcoholic fatty liver disease

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Reduced heart size and increased myocardial fuel substrate oxidation in ACC2 mutant mice

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Continuous fat oxidation in acetyl-CoA carboxylase 2 knockout mice increases total energy expenditure, reduces fat mass, and improves insulin sensitivity

1 reference

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Synthesis and structure-activity relationships of N-{3-[2-(4-alkoxyphenoxy)thiazol-5-yl]-1- methylprop-2-ynyl}carboxy derivatives as selective acetyl-CoA carboxylase 2 inhibitors

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Liver-specific deletion of acetyl-CoA carboxylase 1 reduces hepatic triglyceride accumulation without affecting glucose homeostasis

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Fatty acid regulation of hepatic gene transcription.

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Mutant mice lacking acetyl-CoA carboxylase 1 are embryonically lethal

1 reference

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Glucose and fat metabolism in adipose tissue of acetyl-CoA carboxylase 2 knockout mice

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Role of peroxisome proliferator-activated receptor-gamma in the glucose-sensing apparatus of liver and beta-cells

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Acetyl-CoA carboxylase 2 mutant mice are protected against obesity and diabetes induced by high-fat/high-carbohydrate diets

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Self-excising retroviral vectors encoding the Cre recombinase overcome Cre-mediated cellular toxicity

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Continuous fatty acid oxidation and reduced fat storage in mice lacking acetyl-CoA carboxylase 2

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Nutritional and hormonal regulation of enzymes in fat synthesis: studies of fatty acid synthase and mitochondrial glycerol-3-phosphate acyltransferase gene transcription

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Human acetyl-CoA carboxylase 2. Molecular cloning, characterization, chromosomal mapping, and evidence for two isoforms

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Cloning of human acetyl-CoA carboxylase-beta and its unique features

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Effect of fasting and refeeding on acetyl-CoA carboxylase in rat hindlimb muscle.

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Diet-induced type II diabetes in C57BL/6J mice

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The lipolysis/esterification cycle of hepatic triacylglycerol. Its role in the secretion of very-low-density lipoprotein and its response to hormones and sulphonylureas

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Acute or chronic upregulation of mitochondrial fatty acid oxidation has no net effect on whole-body energy expenditure or adiposity

1 reference

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The life of lipid droplets

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RIP-Cre revisited, evidence for impairments of pancreatic beta-cell function

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Molecular mediators of hepatic steatosis and liver injury

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CP-346086: an MTP inhibitor that lowers plasma cholesterol and triglycerides in experimental animals and in humans

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Heterogeneous metabolic adaptation of C57BL/6J mice to high-fat diet

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Identifiers

10.1074/JBC.M111.309559

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24 January 2020

1 reference

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24 January 2020

1 reference

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24 January 2020

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