Manganese porphyrin reduces renal injury and mitochondrial damage during ischemia/reperfusion. (Q35901001)

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29 December 2019

title

Manganese porphyrin reduces renal injury and mitochondrial damage during ischemia/reperfusion (English)

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29 December 2019

3

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Cheryl Lichti

5

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29 December 2019

2

Ines Batinic-Haberle

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author name string

Hamida Saba

1

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Tanecia Mitchell

4

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29 December 2019

Judit Megyesi

6

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Lee Ann MacMillan-Crow

7

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29 December 2019

publication date

28 February 2007

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Free Radical Biology and Medicine

29 December 2019

published in

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volume

42

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29 December 2019

page(s)

1571-1578

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issue

10

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Overexpression of manganese superoxide dismutase protects against ATP depletion-mediated cell death of proximal tubule cells

29 December 2019

cites work

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Mitochondrial targets of oxidative stress during renal ischemia/reperfusion

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Manganese superoxide dismutase mediates the early release of mitochondrial cytochrome C and subsequent DNA fragmentation after permanent focal cerebral ischemia in mice

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Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury

27 July 2018

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Role of oxidants in ischemic brain damage

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27 July 2018

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27 July 2018

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27 July 2018

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Effects of a manganese (III) porphyrin catalytic antioxidant in a mouse closed head injury model

27 July 2018

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Beneficial effects of MnTBAP, a broad-spectrum reactive species scavenger, in rat renal ischemia/reperfusion injury.

24 September 2018

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Manganese porphyrin given at symptom onset markedly extends survival of ALS mice

24 September 2018

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24 September 2018

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Septic diaphragmatic dysfunction is prevented by Mn(III)porphyrin therapy and inducible nitric oxide synthase inhibition.

24 September 2018

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24 September 2018

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24 September 2018

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24 September 2018

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24 September 2018

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24 September 2018

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24 September 2018

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24 September 2018

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

10.1016/J.FREERADBIOMED.2007.02.016

Identifiers

DOI

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29 December 2019

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29 December 2019

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