Hyperglycemia Induces Cellular Hypoxia through Production of Mitochondrial ROS Followed by Suppression of Aquaporin-1. (Q36068898)

12 August 2017

Hyperglycemia Induces Cellular Hypoxia through Production of Mitochondrial ROS Followed by Suppression of Aquaporin-1. (English)

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12 August 2017

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Kiminori Sada

1

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12 August 2017

Takeshi Nishikawa

2

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12 August 2017

Daisuke Kukidome

3

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12 August 2017

Tomoaki Yoshinaga

4

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12 August 2017

Nobuhiro Kajihara

5

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12 August 2017

Kazuhiro Sonoda

6

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12 August 2017

Takafumi Senokuchi

7

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12 August 2017

Hiroyuki Motoshima

8

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12 August 2017

Takeshi Matsumura

9

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12 August 2017

Eiichi Araki

10

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12 August 2017

language of work or name

English

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publication date

6 July 2016

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12 August 2017

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12 August 2017

11

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12 August 2017

7

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12 August 2017

e0158619

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Creative Commons Attribution 4.0 International

12 August 2017

start time

6 July 2016

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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describes a project that uses

ImageJ

1 reference

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC4934928/fullTextXML

inferred from PubMed Central ID database lookup

cites work

Aquaporin-1 and sodium-hydrogen exchangers as pharmacological targets in diabetic atherosclerosis

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Effects of the antioxidant drug tempol on renal oxygenation in mice with reduced renal mass

2 October 2017

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Cellular hypoxia of pancreatic beta-cells due to high levels of oxygen consumption for insulin secretion in vitro

2 October 2017

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Phosphorescent light-emitting iridium complexes serve as a hypoxia-sensing probe for tumor imaging in living animals

2 October 2017

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Activation of protein kinase C isoforms and its impact on diabetic complications

2 October 2017

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Vascular complications in diabetes mellitus: the role of endothelial dysfunction

2 October 2017

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Hypoxia--a key regulatory factor in tumour growth

2 October 2017

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2 October 2017

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Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

2 October 2017

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Bcl-2 and mitochondrial oxygen radicals. New approaches with reactive oxygen species-sensitive probes.

2 October 2017

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Hyperglycemic pseudohypoxia and diabetic complications.

2 October 2017

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Evidence that hypoxia markers detect oxygen gradients in liver: pimonidazole and retrograde perfusion of rat liver

2 October 2017

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NA+/H+ exchanger 1- and aquaporin-1-dependent hyperosmolarity changes decrease nitric oxide production and induce VCAM-1 expression in endothelial cells exposed to high glucose.

2 October 2017

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Reductive metabolism of the hypoxia marker pimonidazole is regulated by oxygen tension independent of the pyridine nucleotide redox state

11 July 2018

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Endothelial MnSOD overexpression prevents retinal VEGF expression in diabetic mice.

11 July 2018

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Effects of alpha-lipoic acid on ischemia-reperfusion-induced renal dysfunction in rats

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Activation of AMP-activated protein kinase reduces hyperglycemia-induced mitochondrial reactive oxygen species production and promotes mitochondrial biogenesis in human umbilical vein endothelial cells

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The role of aquaporin-1 (AQP1) expression in a murine model of lipopolysaccharide-induced acute lung injury

23 September 2018

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Evaluation of Urinary 8-Hydroxydeoxy-Guanosine as a Novel Biomarker of Macrovascular Complications in Type 2 Diabetes

23 September 2018

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Permeability of lipid membranes to dioxygen

23 September 2018

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PubMed

https://pubmed.ncbi.nlm.nih.gov/27383386

12 December 2020

inferred from PubMed ID database lookup

Development of Cytosolic Hypoxia and Hypoxia-inducible Factor Stabilization Are Facilitated by Aquaporin-1 Expression

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/27383386

12 December 2020

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Oxidative damage to DNA in diabetes mellitus

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PubMed

https://pubmed.ncbi.nlm.nih.gov/27383386

12 December 2020

inferred from PubMed ID database lookup

Identifiers

DOI

10.1371/JOURNAL.PONE.0158619

1 reference

12 August 2017

0 references

1 reference

12 August 2017

PubMed publication ID

27383386

1 reference