Elevation of highly up-regulated in liver cancer (HULC) by hepatitis B virus X protein promotes hepatoma cell proliferation via down-regulating p18. (Q36122238)

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English	Elevation of highly up-regulated in liver cancer (HULC) by hepatitis B virus X protein promotes hepatoma cell proliferation via down-regulating p18.	scientific article

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4 February 2020

Elevation of highly up-regulated in liver cancer (HULC) by hepatitis B virus X protein promotes hepatoma cell proliferation via down-regulating p18 (English)

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Hepatitis B virus

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hepatocellular carcinoma

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3

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Yumei Du

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Guangyao Kong

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Shuai Zhang

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Tao Zhang

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Yuen Gao

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Lihong Ye

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Xiaodong Zhang

8

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8 June 2012

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10

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Journal of Biological Chemistry

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26302-26311

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4 February 2020

The functional role of long non-coding RNA in human carcinomas

1 reference

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miR-520b regulates migration of breast cancer cells by targeting hepatitis B X-interacting protein and interleukin-8

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The X protein of hepatitis B virus inhibits apoptosis in hepatoma cells through enhancing the methionine adenosyltransferase 2A gene expression and reducing S-adenosylmethionine production

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CREB up-regulates long non-coding RNA, HULC expression through interaction with microRNA-372 in liver cancer

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Hepatitis B virus X protein promotes liver cell proliferation via a positive cascade loop involving arachidonic acid metabolism and p-ERK1/2.

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Hepatitis B virus X protein affects S phase progression leading to chromosome segregation defects by binding to damaged DNA binding protein 1.

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Characterization of HULC, a novel gene with striking up-regulation in hepatocellular carcinoma, as noncoding RNA.

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Effects of hepatitis B virus X protein on the development of liver cancer

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The haploinsufficient tumor suppressor p18 upregulates p53 via interactions with ATM/ATR

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The enigmatic X gene of hepatitis B virus

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23 September 2018

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10.1074/JBC.M112.342113

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