Reduced Apaf-1 levels in cardiomyocytes engage strict regulation of apoptosis by endogenous XIAP. (Q36320565)

13 August 2017

Reduced Apaf-1 levels in cardiomyocytes engage strict regulation of apoptosis by endogenous XIAP. (English)

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13 August 2017

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Malia B Potts

1

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13 August 2017

Allyson E Vaughn

2

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13 August 2017

Holly McDonough

3

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13 August 2017

Cam Patterson

4

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13 August 2017

Mohanish Deshmukh

5

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13 August 2017

language of work or name

English

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publication date

12 December 2005

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13 August 2017

Journal of Cell Biology

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13 August 2017

171

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6

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13 August 2017

925-930

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describes a project that uses

13 August 2017

ImageQuant

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC2171313/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

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Apoptosis and heart failure: clinical relevance and therapeutic target

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Decreased apoptosome activity with neuronal differentiation sets the threshold for strict IAP regulation of apoptosis

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Critical function of endogenous XIAP in regulating caspase activation during sympathetic neuronal apoptosis

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Regulation of cardiac myocyte cell death

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IAP proteins: blocking the road to death's door

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Apoptosis in heart failure: release of cytochrome c from mitochondria and activation of caspase-3 in human cardiomyopathy

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Role of cytochrome c and dATP/ATP hydrolysis in Apaf-1-mediated caspase-9 activation and apoptosis

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Apoptosis induced by microinjection of cytochrome c is caspase-dependent and is inhibited by Bcl-2.

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IAP family proteins--suppressors of apoptosis

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Changes in E2F complexes containing retinoblastoma protein family members and increased cyclin-dependent kinase inhibitor activities during terminal differentiation of cardiomyocytes

30 September 2017

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Cell-specific induction of apoptosis by microinjection of cytochrome c. Bcl-xL has activity independent of cytochrome c release

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Apoptotic pathway activation from mitochondria and death receptors without caspase-3 cleavage in failing human myocardium: fragile balance of myocyte survival?

30 September 2017

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Blocking cytochrome c activity within intact neurons inhibits apoptosis.

29 June 2018

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Lack of Apaf-1 expression confers resistance to cytochrome c-driven apoptosis in cardiomyocytes

29 June 2018

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Exogenous smac induces competence and permits caspase activation in sympathetic neurons.

22 September 2018

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Differential expression of apoptotic protease-activating factor-1 and caspase-3 genes and susceptibility to apoptosis during brain development and after traumatic brain injury.

22 September 2018

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Apaf-1 is a transcriptional target for E2F and p53

22 September 2018

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Crossref

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Evidence of a novel event during neuronal death: development of competence-to-die in response to cytoplasmic cytochrome c

21 January 2018

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PubMed

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12 December 2020

based on heuristic

Human skeletal muscle cytosols are refractory to cytochrome c-dependent activation of type-II caspases and lack APAF-1

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/16344307

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1083/JCB.200504082

1 reference

13 August 2017

1 reference

13 August 2017

1 reference