Reactive oxygen species mediate crosstalk between NF-kappaB and JNK. (Q36337991)

13 August 2017

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title

Reactive oxygen species mediate crosstalk between NF-kappaB and JNK. (English)

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13 August 2017

reactive oxygen species

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Nakano H

1

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13 August 2017

Nakajima A

2

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13 August 2017

Sakon-Komazawa S

3

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13 August 2017

Piao JH

4

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13 August 2017

Xue X

5

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13 August 2017

Okumura K

6

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13 August 2017

1 May 2006

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Cell Death & Differentiation

13 August 2017

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13 August 2017

13

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13 August 2017

5

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13 August 2017

730-737

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4401830

13 August 2017

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Correlation between sustained c-Jun N-terminal protein kinase activation and apoptosis induced by tumor necrosis factor-alpha in rat mesangial cells.

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Activation of the JNK signaling pathway: breaking the brake on apoptosis

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Tumor necrosis factor: an apoptosis JuNKie?

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The Jnk1 and Jnk2 protein kinases are required for regional specific apoptosis during early brain development.

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Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

21 January 2018

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MKK7 couples stress signalling to G2/M cell-cycle progression and cellular senescence.

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Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

21 January 2018

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Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

21 January 2018

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ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

21 January 2018

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Bcl-2 undergoes phosphorylation by c-Jun N-terminal kinase/stress-activated protein kinases in the presence of the constitutively active GTP-binding protein Rac1.

21 January 2018

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Bcl-xL blocks activation of related adhesion focal tyrosine kinase/proline-rich tyrosine kinase 2 and stress-activated protein kinase/c-Jun N-terminal protein kinase in the cellular response to methylmethane sulfonate

21 January 2018

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BCL-2 is phosphorylated and inactivated by an ASK1/Jun N-terminal protein kinase pathway normally activated at G(2)/M.

21 January 2018

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Novel role for JNK as a stress-activated Bcl2 kinase

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The Bax subfamily of Bcl2-related proteins is essential for apoptotic signal transduction by c-Jun NH(2)-terminal kinase

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JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis

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JNK promotes Bax translocation to mitochondria through phosphorylation of 14-3-3 proteins

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Stress-signalling kinase Sek1 protects thymocytes from apoptosis mediated by CD95 and CD3

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MEKK1 suppresses oxidative stress-induced apoptosis of embryonic stem cell-derived cardiac myocytes

21 January 2018

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JunD mediates survival signaling by the JNK signal transduction pathway.

21 January 2018

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JNK suppresses apoptosis via phosphorylation of the proapoptotic Bcl-2 family protein BAD

21 January 2018

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Active repression of antiapoptotic gene expression by RelA(p65) NF-kappa B.

21 January 2018

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Inhibition of JNK activation through NF-kappaB target genes

21 January 2018

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Induction of gadd45beta by NF-kappaB downregulates pro-apoptotic JNK signalling.

21 January 2018

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A family of stress-inducible GADD45-like proteins mediate activation of the stress-responsive MTK1/MEKK4 MAPKKK

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X-linked IAP is a direct inhibitor of cell-death proteases

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Gadd45 beta mediates the NF-kappa B suppression of JNK signalling by targeting MKK7/JNKK2

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NF-kappaB signaling pathways in mammalian and insect innate immunity

21 January 2018

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Targeting of TAK1 by the NF-kappa B protein Relish regulates the JNK-mediated immune response in Drosophila

21 January 2018

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The ubiquitin ligase SCFFbw7 antagonizes apoptotic JNK signaling

21 January 2018

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Human De-etiolated-1 regulates c-Jun by assembling a CUL4A ubiquitin ligase

21 January 2018

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NF-kappaB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell death

21 January 2018

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Signaling crosstalk between NF-kappaB and JNK.

21 January 2018

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Linking JNK signaling to NF-kappaB: a key to survival

21 January 2018

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Mitochondria, oxidants, and aging

21 January 2018

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NOX enzymes and the biology of reactive oxygen

21 January 2018

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Free radicals in the physiological control of cell function

21 January 2018

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JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species

21 January 2018

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Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

21 January 2018

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Critical roles of TRAF2 and TRAF5 in tumor necrosis factor-induced NF-kappa B activation and protection from cell death

21 January 2018

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Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

21 January 2018

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Structure and regulation of MAPK phosphatases

21 January 2018

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MEK kinase 1, a substrate for DEVD-directed caspases, is involved in genotoxin-induced apoptosis

21 January 2018

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21 January 2018

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Activators and target genes of Rel/NF-kappaB transcription factors

21 January 2018

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Genome wide analysis of TNF-inducible genes reveals that antioxidant enzymes are induced by TNF and responsible for elimination of ROS.

21 January 2018

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Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain

21 January 2018

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Mitochondrial control of cell death

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Disruption of HSP90 function reverts tumor necrosis factor-induced necrosis to apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

21 January 2018

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Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

21 January 2018

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A revival of old players.

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2.

21 January 2018

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Ferritin Heavy Chain Upregulation by NF-κB Inhibits TNFα-Induced Apoptosis by Suppressing Reactive Oxygen Species

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Electron Transfer between Cytochrome c and p66Shc Generates Reactive Oxygen Species that Trigger Mitochondrial Apoptosis

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Cell survival and a Gadd45-factor deficiency

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

7 January 2021

The role of c-Jun N-terminal kinase (JNK) in apoptosis induced by ultraviolet C and gamma radiation. Duration of JNK activation may determine cell death and proliferation

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

7 January 2021

Vinblastine-induced phosphorylation of Bcl-2 and Bcl-XL is mediated by JNK and occurs in parallel with inactivation of the Raf-1/MEK/ERK cascade

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

7 January 2021

A JNK-Dependent Pathway Is Required for TNFα-Induced Apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

7 January 2021

Reactive oxygen species in cell signaling

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

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Degradation of misfolded proteins prevents ER-derived oxidative stress and cell death

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

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More than one way to die: apoptosis, necrosis and reactive oxygen damage

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401830

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Identifiers

DOI

10.1038/SJ.CDD.4401830

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Dimensions Publication ID

13 August 2017

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