The primary mechanism of attenuation of bacillus Calmette-Guerin is a loss of secreted lytic function required for invasion of lung interstitial tissue (Q36350501)

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English	The primary mechanism of attenuation of bacillus Calmette-Guerin is a loss of secreted lytic function required for invasion of lung interstitial tissue	scientific article

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20 December 2019

The primary mechanism of attenuation of bacillus Calmette-Guerin is a loss of secreted lytic function required for invasion of lung interstitial tissue (English)

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20 December 2019

Suzie Hingley-Wilson

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20 December 2019

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20 December 2019

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Tsungda Hsu

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20 December 2019

Bing Chen

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20 December 2019

Mei Chen

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20 December 2019

Annie Z Dai

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20 December 2019

Paul M Morin

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20 December 2019

Carolyn B Marks

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20 December 2019

Jeevan Padiyar

8

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20 December 2019

Celia Goulding

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20 December 2019

Mari Gingery

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20 December 2019

David Eisenberg

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20 December 2019

Robert G Russell

12

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20 December 2019

Steven C Derrick

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20 December 2019

Frank M Collins

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20 December 2019

Sheldon L Morris

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20 December 2019

C Harold King

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20 December 2019

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13 October 2003

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20 December 2019

Proceedings of the National Academy of Sciences of the United States of America

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20 December 2019

100

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20 December 2019

12420-12425

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20 December 2019

21

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20 December 2019

Deletion of RD1 from Mycobacterium tuberculosis mimics bacille Calmette-Guérin attenuation

1 reference

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30 September 2017

Specialized transduction: an efficient method for generating marked and unmarked targeted gene disruptions in Mycobacterium tuberculosis, M. bovis BCG and M. smegmatis

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Characterization of a Mycobacterium tuberculosis H37Rv transposon library reveals insertions in 351 ORFs and mutants with altered virulence

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Conclusive evidence that the major T-cell antigens of the Mycobacterium tuberculosis complex ESAT-6 and CFP-10 form a tight, 1:1 complex and characterization of the structural properties of ESAT-6, CFP-10, and the ESAT-6*CFP-10 complex. Implications

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The efficiency of the translocation of Mycobacterium tuberculosis across a bilayer of epithelial and endothelial cells as a model of the alveolar wall is a consequence of transport within mononuclear phagocytes and invasion of alveolar epithelial ce

1 reference

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30 September 2017

The ESAT-6 gene cluster of Mycobacterium tuberculosis and other high G+C Gram-positive bacteria

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30 September 2017

Genomics of Mycobacterium bovis

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Necrosis of lung epithelial cells during infection with Mycobacterium tuberculosis is preceded by cell permeation

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Complex lipid determines tissue-specific replication of Mycobacterium tuberculosis in mice

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Comparison of the construction of unmarked deletion mutations in Mycobacterium smegmatis, Mycobacterium bovis bacillus Calmette-Guérin, and Mycobacterium tuberculosis H37Rv by allelic exchange.

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12 December 2020

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10.1073/PNAS.1635213100

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20 December 2019

2003PNAS..10012420H

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20 December 2019

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20 December 2019

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