MicroRNA-208a Dysregulates Apoptosis Genes Expression and Promotes Cardiomyocyte Apoptosis during Ischemia and Its Silencing Improves Cardiac Function after Myocardial Infarction. (Q36356613)

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English	MicroRNA-208a Dysregulates Apoptosis Genes Expression and Promotes Cardiomyocyte Apoptosis during Ischemia and Its Silencing Improves Cardiac Function after Myocardial Infarction.	scientific article

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26688617%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

MicroRNA-208a Dysregulates Apoptosis Genes Expression and Promotes Cardiomyocyte Apoptosis during Ischemia and Its Silencing Improves Cardiac Function after Myocardial Infarction (English)

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26688617%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

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apoptotic process

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based on heuristic

inferred from title

3

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3 March 2020

author name string

Hasahya Tony

1

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3 March 2020

Kai Meng

2

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3 March 2020

Aijia Yu

4

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3 March 2020

Qiutang Zeng

5

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3 March 2020

Kunwu Yu

6

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3 March 2020

Yucheng Zhong

7

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3 March 2020

publication date

25 November 2015

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3 March 2020

Mediators of Inflammation

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3 March 2020

2015

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3 March 2020

479123

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PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26688617%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

copyright license

Creative Commons Attribution 3.0 Unported

1 January 2015

1 reference

April 2022 Public Data File from Crossref

copyright status

1 reference

April 2022 Public Data File from Crossref

miR-1, miR-133a/b, and miR-208a in human fetal hearts correlate to the apoptotic and proliferation markers

1 reference

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Regulation of microRNA biogenesis

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30 September 2017

MicroRNA-208a increases myocardial fibrosis via endoglin in volume overloading heart

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30 September 2017

Mechanical stretch via transforming growth factor-β1 activates microRNA208a to regulate endoglin expression in cultured rat cardiac myoblasts

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30 September 2017

Therapeutic inhibition of miR-208a improves cardiac function and survival during heart failure

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30 September 2017

Insulin promotes vascular smooth muscle cell proliferation via microRNA-208-mediated downregulation of p21.

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30 September 2017

miR-24 inhibits apoptosis and represses Bim in mouse cardiomyocytes

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4673358

30 September 2017

MicroRNA-494 targeting both proapoptotic and antiapoptotic proteins protects against ischemia/reperfusion-induced cardiac injury

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4673358

30 September 2017

Expression of microRNA-208 is associated with adverse clinical outcomes in human dilated cardiomyopathy

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30 September 2017

MicroRNAs miR-1, miR-133a, miR-133b and miR-208 are dysregulated in human myocardial infarction

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4673358

30 September 2017

A family of microRNAs encoded by myosin genes governs myosin expression and muscle performance

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4673358

30 September 2017

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4673358

30 September 2017

Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4673358

30 September 2017

Bioinformatics enrichment tools: paths toward the comprehensive functional analysis of large gene lists

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30 September 2017

Mechanisms of Post-Infarct Left Ventricular Remodeling

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4673358

30 September 2017

MicroRNA-373 induces expression of genes with complementary promoter sequences

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30 September 2017

Switching from repression to activation: microRNAs can up-regulate translation

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30 September 2017

Reciprocal Effects of Micro-RNA-122 on Expression of Heme Oxygenase-1 and Hepatitis C Virus Genes in Human Hepatocytes

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30 September 2017

Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice

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30 September 2017

Control of stress-dependent cardiac growth and gene expression by a microRNA

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30 September 2017

How the Bcl-2 family of proteins interact to regulate apoptosis.

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30 September 2017

Fibrosis as a therapeutic target post-myocardial infarction.

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The mitochondrial death pathway and cardiac myocyte apoptosis

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30 September 2017

Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity

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30 September 2017

The role of the cyclin-dependent kinase inhibitor p21 in apoptosis.

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30 September 2017

Left ventricular remodeling after myocardial infarction: pathophysiology and therapy

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MicroRNA-24 regulates cardiac fibrosis after myocardial infarction

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29 June 2018

MicroRNA: Biogenesis, Function and Role in Cancer

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Cardiac fibroblasts regulate myocardial proliferation through beta1 integrin signaling

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22 September 2018

Cardiac myocyte apoptosis provokes adverse cardiac remodeling in transgenic mice with targeted TNF overexpression.

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Different roles of PPAR-γ activity on physiological and pathological alteration after myocardial ischemia

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Cardiomyocyte apoptosis and ventricular remodeling after myocardial infarction in rats

1 reference

https://pubmed.ncbi.nlm.nih.gov/26688617

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1155/2015/479123

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26688617%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

PMC publication ID

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Europe PubMed Central

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3 March 2020

PubMed publication ID

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3 March 2020

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