Disrupted-in-schizophrenia-1 Gln31Leu polymorphism results in social anhedonia associated with monoaminergic imbalance and reduction of CREB and β-arrestin-1,2 in the nucleus accumbens in a mouse model of depression. (Q36540020)

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scientific article published on 26 September 2012

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English	Disrupted-in-schizophrenia-1 Gln31Leu polymorphism results in social anhedonia associated with monoaminergic imbalance and reduction of CREB and β-arrestin-1,2 in the nucleus accumbens in a mouse model of depression.	scientific article published on 26 September 2012

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9 March 2020

Disrupted-in-schizophrenia-1 Gln31Leu polymorphism results in social anhedonia associated with monoaminergic imbalance and reduction of CREB and β-arrestin-1,2 in the nucleus accumbens in a mouse model of depression (English)

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9 March 2020

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nucleus accumbens

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Albert Hung Choy Wong

4

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Tatiana V Lipina

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Paul J Fletcher

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Frankie H Lee

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John C Roder

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26 September 2012

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Neuropsychopharmacology

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423-436

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9 March 2020

DISC1 variants 37W and 607F disrupt its nuclear targeting and regulatory role in ATF4-mediated transcription

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β-arrestin-biased signaling by the β-adrenergic receptors

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Reconsidering anhedonia in depression: lessons from translational neuroscience

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Atypical depression: a comprehensive review

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Anhedonia in schizophrenia and major depression: state or trait?

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Emotional side-effects of selective serotonin reuptake inhibitors: qualitative study

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Molecular adaptations underlying susceptibility and resistance to social defeat in brain reward regions

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