Multi-locus genome-wide association analysis supports the role of glutamatergic synaptic transmission in the etiology of major depressive disorder. (Q36591241)

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scientific article published on 13 November 2012

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English	Multi-locus genome-wide association analysis supports the role of glutamatergic synaptic transmission in the etiology of major depressive disorder.	scientific article published on 13 November 2012

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17 February 2020

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Multi-locus genome-wide association analysis supports the role of glutamatergic synaptic transmission in the etiology of major depressive disorder (English)

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genome-wide association study

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P H Lee

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J-Y Jung

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Translational Psychiatry

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17 February 2020

INRICH: interval-based enrichment analysis for genome-wide association studies

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A mega-analysis of genome-wide association studies for major depressive disorder

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Altered expression of synapse and glutamate related genes in post-mortem hippocampus of depressed subjects

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Functional gene group analysis identifies synaptic gene groups as risk factor for schizophrenia.

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A new lead from genetic studies in depressed siblings: assessing studies of chromosome 3.

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Altered expression of glutamate signaling, growth factor, and glia genes in the locus coeruleus of patients with major depression

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Poor replication of candidate genes for major depressive disorder using genome-wide association data

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Autism genome-wide copy number variation reveals ubiquitin and neuronal genes

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