Novel zinc-responsive post-transcriptional mechanisms reciprocally regulate expression of the mouse Slc39a4 and Slc39a5 zinc transporters (Zip4 and Zip5). (Q36647957)

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scientific article published on December 2007

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English	Novel zinc-responsive post-transcriptional mechanisms reciprocally regulate expression of the mouse Slc39a4 and Slc39a5 zinc transporters (Zip4 and Zip5).	scientific article published on December 2007

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scholarly article

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26 December 2019

Novel zinc-responsive post-transcriptional mechanisms reciprocally regulate expression of the mouse Slc39a4 and Slc39a5 zinc transporters (Zip4 and Zip5) (English)

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26 December 2019

Benjamin P Weaver

1

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26 December 2019

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Jodi Dufner-Beattie

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26 December 2019

Taiho Kambe

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26 December 2019

Glen K Andrews

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26 December 2019

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1 December 2007

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26 December 2019

Biological Chemistry

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26 December 2019

388

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26 December 2019

1301-1312

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26 December 2019

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26 December 2019

describes a project that uses

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The mouse acrodermatitis enteropathica gene Slc39a4 (Zip4) is essential for early development and heterozygosity causes hypersensitivity to zinc deficiency

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A histidine-rich cluster mediates the ubiquitination and degradation of the human zinc transporter, hZIP4, and protects against zinc cytotoxicity.

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Mouse ZIP1 and ZIP3 genes together are essential for adaptation to dietary zinc deficiency during pregnancy

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The acrodermatitis enteropathica gene ZIP4 encodes a tissue-specific, zinc-regulated zinc transporter in mice

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A novel member of a zinc transporter family is defective in acrodermatitis enteropathica

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Interrelationships of key variables of human zinc homeostasis: relevance to dietary zinc requirements

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12 December 2020

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Identifiers

10.1515/BC.2007.149

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Europe PubMed Central

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26 December 2019

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26 December 2019

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Europe PubMed Central

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26 December 2019

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