Hypoxia and reoxygenation: a possible mechanism for placental oxidative stress in preeclampsia (Q36686796)

15 August 2017

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title

Hypoxia and reoxygenation: a possible mechanism for placental oxidative stress in preeclampsia (English)

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15 August 2017

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Tai-Ho Hung

1

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15 August 2017

Graham J Burton

series ordinal

2

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15 August 2017

publication date

1 September 2006

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Taiwanese Journal of Obstetrics & Gynecology

15 August 2017

published in

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15 August 2017

volume

45

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15 August 2017

issue

3

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15 August 2017

page(s)

189-200

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Calcium supplementation during pregnancy: a systematic review of randomised controlled trials

15 August 2017

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Pathophysiology of hypertension during preeclampsia linking placental ischemia with endothelial dysfunction

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7 January 2021

Expression of the placental cytokines tumor necrosis factor alpha, interleukin 1beta, and interleukin 10 is increased in preeclampsia.

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7 January 2021

TNF alpha concentrations and mRNA expression are increased in preeclamptic placentas

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Tumor necrosis factor-alpha-induced hypertension in pregnant rats results in decreased renal neuronal nitric oxide synthase expression

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TNF-alpha enhances contraction and inhibits endothelial NO-cGMP relaxation in systemic vessels of pregnant rats

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In vitro ischemia-reperfusion injury in term human placenta as a model for oxidative stress in pathological pregnancies

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Enhanced protein levels of protein thiol/disulphide oxidoreductases in placentae from pre-eclamptic subjects

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7 January 2021

Hypoxia-reoxygenation: a potent inducer of apoptotic changes in the human placenta and possible etiological factor in preeclampsia.

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Secretion of tumor necrosis factor-alpha from human placental tissues induced by hypoxia-reoxygenation causes endothelial cell activation in vitro: a potential mediator of the inflammatory response in preeclampsia.

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7 January 2021

10.1016/S1028-4559(09)60224-2

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