A triple entente: virus, neurons, and CD8+ T cells maintain HSV-1 latency (Q36751602)

16 August 2017

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title

A triple entente: virus, neurons, and CD8+ T cells maintain HSV-1 latency (English)

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Herpes simplex virus type 1

16 August 2017

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Sherrie Divito

1

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16 August 2017

Thomas L Cherpes

series ordinal

2

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16 August 2017

Robert L Hendricks

series ordinal

3

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16 August 2017

language of work or name

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1 January 2006

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16 August 2017

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16 August 2017

36

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16 August 2017

1-3

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16 August 2017

119-126

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https://scigraph.springernature.com/pub.10.1385/ir:36:1:119

16 August 2017

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cites work

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12 December 2020

Regulation of caspase 8- and caspase 9-induced apoptosis by the herpes simplex virus type 1 latency-associated transcript

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12 December 2020

Herpes Simplex Virus-Specific Memory CD8+ T Cells Are Selectively Activated and Retained in Latently Infected Sensory Ganglia

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Tracking the spread of a lacZ-tagged herpes simplex virus type 1 between the eye and the nervous system of the mouse: comparison of primary and recurrent infection

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The herpes simplex virus 1 protein kinase US3 is required for protection from apoptosis induced by the virus

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HSV and glycoprotein J inhibit caspase activation and apoptosis induced by granzyme B or Fas.

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Acyclovir blocks cytokine gene expression in trigeminal ganglia latently infected with herpes simplex virus type 1.

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Neurokinin 1 receptor signaling affects the local innate immune defense against genital herpes virus infection

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Neuropeptides, by direct interaction with T cells, induce cytokine secretion and break the commitment to a distinct T helper phenotype

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Gamma interferon expression during acute and latent nervous system infection by herpes simplex virus type 1.

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12 December 2020

Gamma interferon can block herpes simplex virus type 1 reactivation from latency, even in the presence of late gene expression

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Stress-associated immunomodulation and herpes simplex virus infections

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Latent herpesvirus infection in human trigeminal ganglia causes chronic immune response

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Analysis of the cytolytic T-lymphocyte response to herpes simplex virus type 1 glycoprotein B during primary and secondary infection

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Characterization of diverse primary herpes simplex virus type 1 gB-specific cytotoxic T-cell response showing a preferential V beta bias

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12 December 2020

Persistent elevated expression of cytokine transcripts in ganglia latently infected with herpes simplex virus in the absence of ganglionic replication or reactivation

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12 December 2020

Cytokine and chemokine production in HSV-1 latently infected trigeminal ganglion cell cultures: effects of hyperthermic stress.

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12 December 2020

Latent infection with herpes simplex virus is associated with ongoing CD8+ T-cell stimulation by parenchymal cells within sensory ganglia

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Engagement of T cell receptor triggers its recruitment to low-density detergent-insoluble membrane domains

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12 December 2020

An analysis of herpes simplex virus gene expression during latency establishment and reactivation

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12 December 2020

The probability of in vivo reactivation of herpes simplex virus type 1 increases with the number of latently infected neurons in the ganglia

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12 December 2020

Herpes simplex virus inhibits apoptosis through the action of two genes, Us5 and Us3.

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12 December 2020

Effect of neuropeptides on production of inflammatory cytokines by human monocytes

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12 December 2020

The murine double-stranded RNA-dependent protein kinase PKR and the murine 2',5'-oligoadenylate synthetase-dependent RNase L are required for IFN-beta-mediated resistance against herpes simplex virus type 1 in primary trigeminal ganglion culture

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12 December 2020

Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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12 December 2020

Pathways of viral gene expression during acute neuronal infection with HSV-1

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12 December 2020

CD8(+) T cells can block herpes simplex virus type 1 (HSV-1) reactivation from latency in sensory neurons

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12 December 2020

Qa-1b and CD94-NKG2a interaction regulate cytolytic activity of herpes simplex virus-specific memory CD8+ T cells in the latently infected trigeminal ganglia

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12 December 2020

Reduced CTL response and increased viral burden in substance P receptor-deficient mice infected with murine gamma-herpesvirus 68

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12 December 2020

The latent herpes simplex virus type 1 genome copy number in individual neurons is virus strain specific and correlates with reactivation

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12 December 2020

Immune control of HSV-1 latency

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12 December 2020

Restricted herpes simplex virus type 1 gene expression within sensory neurons in the absence of functional B and T lymphocytes

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12 December 2020

Persistent cytokine expression in trigeminal ganglion latently infected with herpes simplex virus type 1.

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12 December 2020

Neurotransmitters regulate the migration and cytotoxicity in natural killer cells

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Relationship of herpes simplex virus genome configuration to productive and persistent infections

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Substance P: a pioneer amongst neuropeptides

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12 December 2020

The gene that encodes the herpes simplex virus type 1 latency-associated transcript influences the accumulation of transcripts (Bcl-x(L) and Bcl-x(S)) that encode apoptotic regulatory proteins.

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Substance P increases in vitro lymphokine-activated-killer (LAK) cell cytotoxicity against fresh colorectal cancer cells

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Severe genital herpes infections in HIV-infected individuals with impaired herpes simplex virus-specific CD8+ cytotoxic T lymphocyte responses

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12 December 2020

Anti-CD8 impairs clearance of herpes simplex virus from the nervous system: implications for the fate of virally infected neurons

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12 December 2020

Identifiers

DOI

10.1385/IR:36:1:119

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Dimensions Publication ID

16 August 2017

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