STAT3 activity is necessary and sufficient for the development of immune-mediated myocarditis in mice and promotes progression to dilated cardiomyopathy. (Q36771328)

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scientific article published on 5 March 2013

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English	STAT3 activity is necessary and sufficient for the development of immune-mediated myocarditis in mice and promotes progression to dilated cardiomyopathy.	scientific article published on 5 March 2013

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25 February 2020

STAT3 activity is necessary and sufficient for the development of immune-mediated myocarditis in mice and promotes progression to dilated cardiomyopathy (English)

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25 February 2020

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25 February 2020

Roberto Chiarle

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25 February 2020

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Annalisa Camporeale

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25 February 2020

Francesca Marino

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Anna Papageorgiou

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25 February 2020

Sara Fornero

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25 February 2020

Steven Fletcher

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25 February 2020

Brent D G Page

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25 February 2020

Patrick Gunning

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25 February 2020

Marco Forni

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25 February 2020

Mara Morello

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25 February 2020

Ole Jensen

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25 February 2020

Renzo Levi

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25 February 2020

Stephane Heymans

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25 February 2020

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5 March 2013

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25 February 2020

EMBO Molecular Medicine

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25 February 2020

5

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25 February 2020

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25 February 2020

572-590

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25 February 2020

describes a project that uses

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3628107/fullTextXML

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IL-6, IL-17 and STAT3: a holy trinity in auto-immunity?

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Complement regulation of T cell immunity

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29 September 2017

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Absence of SOCS3 in the cardiomyocyte increases mortality in a gp130-dependent manner accompanied by contractile dysfunction and ventricular arrhythmias

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29 September 2017

The management of myocarditis

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29 September 2017

Myocarditis: infection versus autoimmunity

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29 September 2017

Pathogenesis of myocarditis and dilated cardiomyopathy

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STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

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29 September 2017

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Identification of cardiac myosin peptides capable of inducing autoimmune myocarditis in BALB/c mice

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5 September 2018

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Continuous Glycoprotein-130–Mediated Signal Transducer and Activator of Transcription-3 Activation Promotes Inflammation, Left Ventricular Rupture, and Adverse Outcome in Subacute Myocardial Infarction

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Interleukin-17A Is Dispensable for Myocarditis but Essential for the Progression to Dilated Cardiomyopathy

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1 reference

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1 reference

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1 reference

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1 reference

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12 December 2020

based on heuristic

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IL-6-gp130-STAT3 in T cells directs the development of IL-17+ Th with a minimum effect on that of Treg in the steady state

1 reference

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1 reference

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1 reference

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Identifiers

10.1002/EMMM.201201876

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25 February 2020

PMC publication ID

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Europe PubMed Central

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25 February 2020

PubMed publication ID

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Europe PubMed Central

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25 February 2020

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