Modulation of SR Ca release by luminal Ca and calsequestrin in cardiac myocytes: effects of CASQ2 mutations linked to sudden cardiac death. (Q36791253)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18469084%20AND%20SRC:MED&resulttype=core&format=json

7 January 2020

title

Modulation of SR Ca release by luminal Ca and calsequestrin in cardiac myocytes: effects of CASQ2 mutations linked to sudden cardiac death (English)

1 reference

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7 January 2020

4

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7 January 2020

Donald M. Bers

8

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7 January 2020

author name string

Dmitry Terentyev

1

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7 January 2020

Zuzana Kubalova

2

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7 January 2020

Giorgia Valle

3

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7 January 2020

Srikanth Vedamoorthyrao

5

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7 January 2020

Radmila Terentyeva

6

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7 January 2020

Serge Viatchenko-Karpinski

7

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7 January 2020

Simon C Williams

9

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7 January 2020

Pompeo Volpe

10

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7 January 2020

Sandor Gyorke

11

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7 January 2020

language of work or name

English

0 references

publication date

9 May 2008

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7 January 2020

number of pages

12

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based on heuristic

inferred from page(s)

published in

Biophysical Journal

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7 January 2020

volume

95

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7 January 2020

issue

4

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7 January 2020

page(s)

2037-2048

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Partial inhibition of sarcoplasmic reticulum ca release evokes long-lasting ca release events in ventricular myocytes: role of luminal ca in termination of ca release

7 January 2020

cites work

1 reference

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Arrhythmogenic mechanisms in a mouse model of catecholaminergic polymorphic ventricular tachycardia

29 September 2017

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Calsequestrin 2 (CASQ2) mutations increase expression of calreticulin and ryanodine receptors, causing catecholaminergic polymorphic ventricular tachycardia

29 September 2017

1 reference

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Casq2 deletion causes sarcoplasmic reticulum volume increase, premature Ca2+ release, and catecholaminergic polymorphic ventricular tachycardia

29 September 2017

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Clinical phenotype and functional characterization of CASQ2 mutations associated with catecholaminergic polymorphic ventricular tachycardia

29 September 2017

1 reference

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Abnormal interactions of calsequestrin with the ryanodine receptor calcium release channel complex linked to exercise-induced sudden cardiac death

29 September 2017

1 reference

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Abnormal intrastore calcium signaling in chronic heart failure

29 September 2017

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Ca2+ blinks: rapid nanoscopic store calcium signaling

29 September 2017

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Calsequestrin mutant D307H exhibits depressed binding to its protein targets and a depressed response to calcium

29 September 2017

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Sarcoplasmic reticulum Ca2+ refilling controls recovery from Ca2+-induced Ca2+ release refractoriness in heart muscle

29 September 2017

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Calsequestrin and the calcium release channel of skeletal and cardiac muscle

29 September 2017

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The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium

29 September 2017

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Protein measurement with the Folin phenol reagent

29 September 2017

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Comparing skeletal and cardiac calsequestrin structures and their calcium binding: a proposed mechanism for coupled calcium binding and protein polymerization

29 September 2017

1 reference

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Abnormal calcium signaling and sudden cardiac death associated with mutation of calsequestrin

29 September 2017

1 reference

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Calsequestrin determines the functional size and stability of cardiac intracellular calcium stores: Mechanism for hereditary arrhythmia

29 September 2017

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Termination of cardiac Ca(2+) sparks: an investigative mathematical model of calcium-induced calcium release

29 September 2017

1 reference

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A missense mutation in a highly conserved region of CASQ2 is associated with autosomal recessive catecholamine-induced polymorphic ventricular tachycardia in Bedouin families from Israel

29 September 2017

1 reference

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The asp-rich region at the carboxyl-terminus of calsequestrin binds to Ca(2+) and interacts with triadin

29 September 2017

1 reference

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Termination of Ca2+ release by a local inactivation of ryanodine receptors in cardiac myocytes

29 September 2017

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Crystal structure of calsequestrin from rabbit skeletal muscle sarcoplasmic reticulum

29 September 2017

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Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane

29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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Modulation of cytosolic and intra-sarcoplasmic reticulum calcium waves by calsequestrin in rat cardiac myocytes

29 September 2017

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Termination of Ca2+ release during Ca2+ sparks in rat ventricular myocytes.

27 June 2018

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27 June 2018

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Effects of cytosolic NADH/NAD(+) levels on sarcoplasmic reticulum Ca(2+) release in permeabilized rat ventricular myocytes

27 June 2018

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Ca2+ scraps: local depletions of free [Ca2+] in cardiac sarcoplasmic reticulum during contractions leave substantial Ca2+ reserve

5 September 2018

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Luminal Ca2+ controls termination and refractory behavior of Ca2+-induced Ca2+ release in cardiac myocytes

5 September 2018

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Head-to-tail oligomerization of calsequestrin: a novel mechanism for heterogeneous distribution of endoplasmic reticulum luminal proteins

5 September 2018

1 reference

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Intra-sarcoplasmic reticulum free [Ca2+] and buffering in arrhythmogenic failing rabbit heart

5 September 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/18469084

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Absence of calsequestrin 2 causes severe forms of catecholaminergic polymorphic ventricular tachycardia

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/18469084

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1529/BIOPHYSJ.107.128249

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18469084%20AND%20SRC:MED&resulttype=core&format=json

7 January 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18469084%20AND%20SRC:MED&resulttype=core&format=json

7 January 2020

1 reference