Consequences of lipid droplet coat protein downregulation in liver cells: abnormal lipid droplet metabolism and induction of insulin resistance. (Q36807686)

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scientific article published on 16 May 2008

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English	Consequences of lipid droplet coat protein downregulation in liver cells: abnormal lipid droplet metabolism and induction of insulin resistance.	scientific article published on 16 May 2008

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scholarly article

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7 January 2020

Consequences of lipid droplet coat protein downregulation in liver cells: abnormal lipid droplet metabolism and induction of insulin resistance (English)

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insulin resistance

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Michael J. Quon

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Michael J. Quon

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Ming Bell

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Hong Wang

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Hui Chen

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John C McLenithan

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Da-Wei Gong

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Rong-Zee Yang

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Daozhan Yu

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Constantine Londos

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Carole Sztalryd

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16 May 2008

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2037-2045

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describes a project that uses

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Reduction of hepatosteatosis and lipid levels by an adipose differentiation-related protein antisense oligonucleotide.

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OXPAT/PAT-1 is a PPAR-induced lipid droplet protein that promotes fatty acid utilization

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Adipose triglyceride lipase-mediated lipolysis of cellular fat stores is activated by CGI-58 and defective in Chanarin-Dorfman Syndrome

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Adipocyte differentiation-related protein promotes fatty acid storage in cytosolic triglycerides and inhibits secretion of very low-density lipoproteins

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PPARalpha activators and fasting induce the expression of adipose differentiation-related protein in liver

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Protection against fatty liver but normal adipogenesis in mice lacking adipose differentiation-related protein

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Adipose triglyceride lipase: function, regulation by insulin, and comparison with adiponutrin

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The metabolic syndrome

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PPARgamma2 regulates lipogenesis and lipid accumulation in steatotic hepatocytes.

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PKC-theta knockout mice are protected from fat-induced insulin resistance

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Inhibition of insulin sensitivity by free fatty acids requires activation of multiple serine kinases in 3T3-L1 adipocytes

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29 September 2017

CGI-58 interacts with perilipin and is localized to lipid droplets. Possible involvement of CGI-58 mislocalization in Chanarin-Dorfman syndrome

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A RAPID METHOD OF TOTAL LIPID EXTRACTION AND PURIFICATION

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Perilipin A is essential for the translocation of hormone-sensitive lipase during lipolytic activation

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Triglyceride accumulation protects against fatty acid-induced lipotoxicity

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Epidemiology of the metabolic syndrome, 2002.

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Mutations in CGI-58, the gene encoding a new protein of the esterase/lipase/thioesterase subfamily, in Chanarin-Dorfman syndrome

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TIP47 associates with lipid droplets

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Adipocyte differentiation-related protein reduces the lipid droplet association of adipose triglyceride lipase and slows triacylglycerol turnover

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1 reference

https://pubmed.ncbi.nlm.nih.gov/18487449

12 December 2020

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Fractionation of plasma nonesterified fatty acids

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12 December 2020

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10.2337/DB07-1383

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7 January 2020

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7 January 2020

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7 January 2020

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