β-Cell Glucose Sensitivity Is Linked to Insulin/Glucagon Bihormonal Cells in Nondiabetic Humans. (Q36935383)

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2 March 2020

title

β-Cell Glucose Sensitivity Is Linked to Insulin/Glucagon Bihormonal Cells in Nondiabetic Humans (English)

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2 March 2020

1

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Caterina Conte

3

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2 March 2020

author name string

Gian P Sorice

2

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2 March 2020

Vinsin A Sun

4

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2 March 2020

Chiara M A Cefalo

5

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2 March 2020

Simona Moffa

6

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2 March 2020

Alfredo Pontecorvi

7

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2 March 2020

Andrea Mari

8

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2 March 2020

Rohit N Kulkarni

9

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Andrea Giaccari

10

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language of work or name

English

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publication date

9 December 2015

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The Journal of Clinical Endocrinology and Metabolism

2 March 2020

published in

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volume

101

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page(s)

470-475

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2 March 2020

issue

2

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Insulin resistance alters islet morphology in nondiabetic humans

2 March 2020

cites work

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4880124

Modulation of β-cell function: a translational journey from the bench to the bedside

29 September 2017

1 reference

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Type 2 diabetes across generations: from pathophysiology to prevention and management

29 September 2017

1 reference

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Glucose toxicity: the leading actor in the pathogenesis and clinical history of type 2 diabetes - mechanisms and potentials for treatment

29 September 2017

1 reference

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Exendin-4 stimulates both beta-cell replication and neogenesis, resulting in increased beta-cell mass and improved glucose tolerance in diabetic rats

29 September 2017

1 reference

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Glucose clamp technique: a method for quantifying insulin secretion and resistance

29 September 2017

1 reference

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Insulin secretory responses to rising and falling glucose concentrations are delayed in subjects with impaired glucose tolerance

29 September 2017

1 reference

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Estimation of insulin secretion rates from C-peptide levels. Comparison of individual and standard kinetic parameters for C-peptide clearance

27 June 2018

1 reference

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Role of reduced β-cell mass versus impaired β-cell function in the pathogenesis of type 2 diabetes

27 June 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4880124

Impaired beta cell glucose sensitivity rather than inadequate compensation for insulin resistance is the dominant defect in glucose intolerance

4 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4880124

Progressive loss of beta-cell function leads to worsening glucose tolerance in first-degree relatives of subjects with type 2 diabetes.

4 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4880124

4 September 2018

Identifiers

DOI

10.1210/JC.2015-2802

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2 March 2020

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2 March 2020

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