The skeleton: endocrine regulator of phosphate homeostasis (Q37334547)

19 August 2017

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title

The skeleton: endocrine regulator of phosphate homeostasis (English)

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19 August 2017

Máire E Doyle

1

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PubMed ID

19032923

19 August 2017

Suzanne M Jan de Beur

series ordinal

2

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19 August 2017

1 December 2008

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Current osteoporosis reports

19 August 2017

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19 August 2017

6

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19 August 2017

4

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19 August 2017

134-141

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https://scigraph.springernature.com/pub.10.1007/s11914-008-0024-6

19 August 2017

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cites work

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

1 reference

Crossref

https://api.crossref.org/works/10.1007%2FS11914-008-0024-6

The Dentin Matrix Protein 1 (Dmp1) is Specifically Expressed in Mineralized, but not Soft, Tissues during Development

21 January 2018

1 reference

12 December 2020

Circulating FGF-23 is regulated by 1alpha,25-dihydroxyvitamin D3 and phosphorus in vivo

1 reference

12 December 2020

Bone as a source of FGF23: regulation by phosphate?

1 reference

12 December 2020

A novel mutation in fibroblast growth factor 23 gene as a cause of tumoral calcinosis

1 reference

12 December 2020

Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

1 reference

12 December 2020

Klotho is a novel beta-glucuronidase capable of hydrolyzing steroid beta-glucuronides

1 reference

12 December 2020

Vitamin D receptor-independent FGF23 actions in regulating phosphate and vitamin D metabolism

1 reference

12 December 2020

A novel GALNT3 mutation in a pseudoautosomal dominant form of tumoral calcinosis: evidence that the disorder is autosomal recessive

1 reference

12 December 2020

NovelGALNT3Mutations Causing Hyperostosis-Hyperphosphatemia Syndrome Result in Low Intact Fibroblast Growth Factor 23 Concentrations

1 reference

12 December 2020

Mutations in GALNT3, encoding a protein involved in O-linked glycosylation, cause familial tumoral calcinosis

1 reference

12 December 2020

Nephrolithiasis and osteoporosis associated with hypophosphatemia caused by mutations in the type 2a sodium-phosphate cotransporter

1 reference

12 December 2020

In vitro effects of dentin matrix protein-1 on hydroxyapatite formation provide insights into in vivo functions

1 reference

12 December 2020

Mutation of the mouse klotho gene leads to a syndrome resembling ageing

1 reference

12 December 2020

Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice

1 reference

12 December 2020

The parathyroid is a target organ for FGF23 in rats

1 reference

12 December 2020

FGF23 is processed by proprotein convertases but not by PHEX.

1 reference

12 December 2020

Hereditary hypophosphatemic rickets with hypercalciuria

1 reference

12 December 2020

Polypeptide GalNAc-transferase T3 and familial tumoral calcinosis. Secretion of fibroblast growth factor 23 requires O-glycosylation

1 reference

12 December 2020

Novel aspects in regulated expression of the renal type IIa Na/Pi-cotransporter

1 reference

12 December 2020

NHERF1 mutations and responsiveness of renal parathyroid hormone

1 reference

12 December 2020

1alpha,25-Dihydroxyvitamin D3 upregulates FGF23 gene expression in bone: the final link in a renal-gastrointestinal-skeletal axis that controls phosphate transport

1 reference

12 December 2020

DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis.

1 reference

12 December 2020

FGF-23 inhibits renal tubular phosphate transport and is a PHEX substrate

1 reference

12 December 2020

Regulation of fibroblast growth factor-23 signaling by klotho

1 reference

12 December 2020

Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism.

1 reference

12 December 2020

Fibroblast growth factor 23 in oncogenic osteomalacia and X-linked hypophosphatemia

1 reference

12 December 2020

Regulation of fibroblastic growth factor 23 expression but not degradation by PHEX.

1 reference

12 December 2020

An FGF23 missense mutation causes familial tumoral calcinosis with hyperphosphatemia.

1 reference

12 December 2020

Fibroblast growth factor 23 impairs phosphorus and vitamin D metabolism in vivo and suppresses 25-hydroxyvitamin D-1alpha-hydroxylase expression in vitro

1 reference

12 December 2020

Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism

1 reference

12 December 2020

Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23

1 reference

12 December 2020

The roles of specific genes implicated as circulating factors involved in normal and disordered phosphate homeostasis: frizzled related protein-4, matrix extracellular phosphoglycoprotein, and fibroblast growth factor 23.

1 reference

12 December 2020

Inhibition of intestinal sodium-dependent inorganic phosphate transport by fibroblast growth factor 23.

1 reference

12 December 2020

The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting

1 reference

12 December 2020

The autosomal dominant hypophosphatemic rickets R176Q mutation in fibroblast growth factor 23 resists proteolytic cleavage and enhances in vivo biological potency

1 reference

12 December 2020

SLC34A3 mutations in patients with hereditary hypophosphatemic rickets with hypercalciuria predict a key role for the sodium-phosphate cotransporter NaPi-IIc in maintaining phosphate homeostasis

1 reference

12 December 2020

Identification of the human klotho gene and its two transcripts encoding membrane and secreted klotho protein

1 reference

12 December 2020

Intronic deletions in the SLC34A3 gene cause hereditary hypophosphatemic rickets with hypercalciuria

1 reference

12 December 2020

A gene (PEX) with homologies to endopeptidases is mutated in patients with X-linked hypophosphatemic rickets. The HYP Consortium

1 reference

12 December 2020

A novel homozygous missense mutation in FGF23 causes Familial Tumoral Calcinosis associated with disseminated visceral calcification

1 reference

12 December 2020

Mutant FGF-23 responsible for autosomal dominant hypophosphatemic rickets is resistant to proteolytic cleavage and causes hypophosphatemia in vivo.

1 reference

12 December 2020

FGF-23 transgenic mice demonstrate hypophosphatemic rickets with reduced expression of sodium phosphate cotransporter type IIa.

1 reference

12 December 2020

Tumors associated with oncogenic osteomalacia express genes important in bone and mineral metabolism

1 reference

12 December 2020

A translocation causing increased alpha-klotho level results in hypophosphatemic rickets and hyperparathyroidism

1 reference

12 December 2020

Mineralized tissue cells are a principal source of FGF23.

1 reference

12 December 2020

Klotho converts canonical FGF receptor into a specific receptor for FGF23

1 reference

12 December 2020

A homozygous missense mutation in human KLOTHO causes severe tumoral calcinosis

1 reference

12 December 2020

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

1 reference

12 December 2020

Mutations that cause osteoglophonic dysplasia define novel roles for FGFR1 in bone elongation

1 reference

12 December 2020

10.1007/S11914-008-0024-6

Identifiers

DOI

1 reference

19 August 2017

1 reference

PubMed ID

19032923