The role of redox modulation of class II histone deacetylases in mediating pathological cardiac hypertrophy. (Q37474449)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19424677%20AND%20SRC:MED&resulttype=core&format=json

27 January 2020

review article

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title

The role of redox modulation of class II histone deacetylases in mediating pathological cardiac hypertrophy (English)

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27 January 2020

2

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27 January 2020

Junichi Sadoshima

5

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27 January 2020

author name string

Shin-ichi Oka

1

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27 January 2020

Takanari Kitazono

3

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27 January 2020

Daniela Zablocki

4

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27 January 2020

language of work or name

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8 May 2009

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Journal of Molecular Medicine

27 January 2020

published in

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27 January 2020

volume

87

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27 January 2020

issue

8

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27 January 2020

page(s)

785-791

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https://scigraph.springernature.com/pub.10.1007/s00109-009-0471-2

27 January 2020

exact match

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cites work

Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction

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Redox regulation of growth and death in cardiac myocytes

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S-Nitrosylation of histone deacetylase 2 induces chromatin remodelling in neurons

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Role of reactive oxygen species in myocardial remodeling.

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Oxidative stress-induced renal angiotensin AT1 receptor upregulation causes increased stimulation of sodium transporters and hypertension

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Importin alpha1 (Rch1) mediates nuclear translocation of thioredoxin-binding protein-2/vitamin D(3)-up-regulated protein 1

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Human alpha B-crystallin mutation causes oxido-reductive stress and protein aggregation cardiomyopathy in mice

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Protein kinase D mediates mitochondrion-to-nucleus signaling and detoxification from mitochondrial reactive oxygen species

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Histone deacetylases (HDACs): characterization of the classical HDAC family

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The diversity of the DnaJ/Hsp40 family, the crucial partners for Hsp70 chaperones

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Hdac2 regulates the cardiac hypertrophic response by modulating Gsk3 beta activity

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Thioredoxin1 as a negative regulator of cardiac hypertrophy

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Functional significance of histone deacetylase diversity

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An expression screen reveals modulators of class II histone deacetylase phosphorylation

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The transcriptional coactivator CAMTA2 stimulates cardiac growth by opposing class II histone deacetylases

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Activation of histone deacetylase 2 by inducible heat shock protein 70 in cardiac hypertrophy

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Thioredoxin. Overview

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Oxidants painting the cysteine chapel: redox regulation of PTPs

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Txnip balances metabolic and growth signaling via PTEN disulfide reduction

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Local InsP3-dependent perinuclear Ca2+ signaling in cardiac myocyte excitation-transcription coupling

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Identification of thioredoxin-binding protein-2/vitamin D(3) up-regulated protein 1 as a negative regulator of thioredoxin function and expression

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Role of sirtuin proteins in life extension by caloric restriction

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Down-regulation of catalase and oxidative modification of protein kinase CK2 lead to the failure of apoptosis repressor with caspase recruitment domain to inhibit cardiomyocyte hypertrophy

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The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload

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Redox-regulated cochaperone activity of the human DnaJ homolog Hdj2.

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A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation

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The histone deacetylase inhibitor SAHA arrests cancer cell growth, up-regulates thioredoxin-binding protein-2, and down-regulates thioredoxin.

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Redox regulatory and anti-apoptotic functions of thioredoxin depend on S-nitrosylation at cysteine 69.

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Structural and functional analysis of the human HDAC4 catalytic domain reveals a regulatory structural zinc-binding domain

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Protein kinases C and D mediate agonist-dependent cardiac hypertrophy through nuclear export of histone deacetylase 5

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Mitochondrial-nuclear cross-talk in the aging and failing heart

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SIK1 is a class II HDAC kinase that promotes survival of skeletal myocytes

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Reactive oxygen species in mechanical stress-induced cardiac hypertrophy

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12 December 2020

Uncovering G protein-coupled receptor kinase-5 as a histone deacetylase kinase in the nucleus of cardiomyocytes

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Redox-sensitive signaling by angiotensin II involves oxidative inactivation and blunted phosphorylation of protein tyrosine phosphatase SHP-2 in vascular smooth muscle cells from SHR.

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Reactive oxygen species, mitochondria, and NAD(P)H oxidases in the development and progression of heart failure

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Thioredoxin-binding protein-2-like inducible membrane protein is a novel vitamin D3 and peroxisome proliferator-activated receptor (PPAR)gamma ligand target protein that regulates PPARgamma signaling

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Phosphorylation of the respiratory burst oxidase subunit p47phox as determined by two-dimensional phosphopeptide mapping. Phosphorylation by protein kinase C, protein kinase A, and a mitogen-activated protein kinase

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A redox-dependent pathway for regulating class II HDACs and cardiac hypertrophy

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Translating the Histone Code

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12 December 2020

Intravenous allopurinol decreases myocardial oxygen consumption and increases mechanical efficiency in dogs with pacing-induced heart failure

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Downregulation of CuZn-superoxide dismutase contributes to beta-adrenergic receptor-mediated oxidative stress in the heart

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Redox redux: revisiting PTPs and the control of cell signaling

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12 December 2020

Control of cardiac growth by histone acetylation/deacetylation

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12 December 2020

CaMKIIdelta isoforms differentially affect calcium handling but similarly regulate HDAC/MEF2 transcriptional responses

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12 December 2020

10.1007/S00109-009-0471-2

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19424677%20AND%20SRC:MED&resulttype=core&format=json

release_yadyieghzvdthcv6dfehccjpwe

27 January 2020

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Fatcat

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24 November 2022

mapped directly with Wikidata item

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