Herpes simplex virus type 1 persists in the aged brain through hypothetical expression of accessory genes (Q37745039)

22 August 2017

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title

Herpes simplex virus type 1 persists in the aged brain through hypothetical expression of accessory genes (English)

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Herpes simplex virus type 1

22 August 2017

main subject

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Isamu Mori

1

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22 August 2017

publication date

1 May 2010

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22 August 2017

published in

Journal of NeuroVirology

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22 August 2017

volume

16

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22 August 2017

issue

3

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22 August 2017

page(s)

203-207

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Productive herpes simplex virus in brain of elderly normal subjects and Alzheimer's disease patients

22 August 2017

cites work

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CTL are inactivated by herpes simplex virus-infected cells expressing a viral protein kinase

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Stimulation of enveloped virus infection by beta-amyloid fibrils

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Herpes simplex virus protein kinase US3 activates and functionally overlaps protein kinase A to block apoptosis

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Cell surface major histocompatibility complex class II proteins are regulated by the products of the gamma(1)34.5 and U(L)41 genes of herpes simplex virus 1.

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Fibril formation and neurotoxicity by a herpes simplex virus glycoprotein B fragment with homology to the Alzheimer's A beta peptide

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The gamma(1)34.5 protein of herpes simplex virus 1 complexes with protein phosphatase 1alpha to dephosphorylate the alpha subunit of the eukaryotic translation initiation factor 2 and preclude the shutoff of protein synthesis by double-stranded RNA-

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HSV and glycoprotein J inhibit caspase activation and apoptosis induced by granzyme B or Fas.

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Herpes simplex virus type 1 glycoprotein E mediates retrograde spread from epithelial cells to neurites

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Two small RNAs encoded within the first 1.5 kilobases of the herpes simplex virus type 1 latency-associated transcript can inhibit productive infection and cooperate to inhibit apoptosis

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Herpes simplex virus type 1 in brain and risk of Alzheimer's disease

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12 December 2020

Accessory genes define the relationship between the herpes simplex virus and its host

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Basic principles of immunological surveillance of the normal central nervous system

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Herpes simplex virus type 1 Us3 gene deletion influences toll-like receptor responses in cultured monocytic cells

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Herpes simplex virus type 1 evades the effects of antibody and complement in vivo

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Mild forms of herpes encephalitis

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PKR-dependent autophagic degradation of herpes simplex virus type 1.

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Herpes simplex virus type 1 latency-associated transcript inhibits apoptosis and promotes neurite sprouting in neuroblastoma cells following serum starvation by maintaining protein kinase B (AKT) levels

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Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.

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Anterograde spread of herpes simplex virus type 1 requires glycoprotein E and glycoprotein I but not Us9.

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Herpes simplex virus type 1 DNA is located within Alzheimer's disease amyloid plaques

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12 December 2020

The locus encompassing the latency-associated transcript of herpes simplex virus type 1 interferes with and delays interferon expression in productively infected neuroblastoma cells and trigeminal Ganglia of acutely infected mice.

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Olfactory transmission of neurotropic viruses

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Full resistance of herpes simplex virus type 1-infected primary human cells to alpha interferon requires both the Us11 and gamma(1)34.5 gene products.

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12 December 2020

The U(L)41 protein of herpes simplex virus 1 degrades RNA by endonucleolytic cleavage in absence of other cellular or viral proteins

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12 December 2020

Latency of alpha-herpes viruses is accompanied by a chronic inflammation in human trigeminal ganglia but not in dorsal root ganglia.

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12 December 2020

The herpes simplex virus-1 Us3 protein kinase blocks CD8T cell lysis by preventing the cleavage of Bid by granzyme B.

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12 December 2020

HSV ICP0 recruits USP7 to modulate TLR-mediated innate response

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The latency-associated transcript of herpes simplex virus type 1 promotes survival and stimulates axonal regeneration in sympathetic and trigeminal neurons

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12 December 2020

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12 December 2020

Herpes simplex virus gene products: the accessories reflect her lifestyle well

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12 December 2020

The herpes simplex virus ICP0 RING finger domain inhibits IRF3- and IRF7-mediated activation of interferon-stimulated genes

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12 December 2020

Promyelocytic leukemia protein mediates interferon-based anti-herpes simplex virus 1 effects

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12 December 2020

Herpes simplex virus type 1 latency in the murine nervous system is associated with oxidative damage to neurons

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12 December 2020

Expression from the herpes simplex virus type 1 latency-associated promoter in the murine central nervous system

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12 December 2020

Herpes simplex virus type 1 in Alzheimer's disease: the enemy within

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12 December 2020

Interaction of ICP34.5 with Beclin 1 modulates herpes simplex virus type 1 pathogenesis through control of CD4+ T-cell responses

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12 December 2020

Alzheimer's disease-specific tau phosphorylation is induced by herpes simplex virus type 1.

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12 December 2020

The herpes simplex virus type 1 U(S)11 protein interacts with protein kinase R in infected cells and requires a 30-amino-acid sequence adjacent to a kinase substrate domain

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12 December 2020

10.3109/13550281003739040

Identifiers

DOI

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Dimensions Publication ID

22 August 2017

1070985852

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