Clinicopathological characteristics of obesity-associated focal segmental glomerulosclerosis. (Q37886981)

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3 February 2020

review article

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title

Clinicopathological characteristics of obesity-associated focal segmental glomerulosclerosis (English)

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21657818%20AND%20SRC:MED&resulttype=core&format=json

focal segmental glomerulosclerosis

3 February 2020

main subject

obesity

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4

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3 February 2020

author name string

Sihem Darouich

1

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3 February 2020

Rym Goucha

2

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3 February 2020

Mohamed Habib Jaafoura

3

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3 February 2020

Hédi Ben Maiz

5

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3 February 2020

Adel Kheder

6

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publication date

9 June 2011

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Ultrastructural Pathology

3 February 2020

published in

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volume

35

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3 February 2020

page(s)

176-182

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issue

4

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Obesity, proteinuria and progression of renal failure

3 February 2020

cites work

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Clinical features and long-term outcome of obesity-associated focal segmental glomerulosclerosis

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Proteinuria and focal segmental glomerulosclerosis in severely obese adolescents

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Glomerulomegaly and focal segmental glomerulosclerosis associated with obesity and sleep-apnea syndrome

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Obesity-associated focal segmental glomerulosclerosis: pathological features of the lesion and relationship with cardiomegaly and hyperlipidemia

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Podocyte lesions in patients with obesity-related glomerulopathy.

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The renin-angiotensin system in glomerular podocytes: mediator of glomerulosclerosis and link to hypertensive nephropathy

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NADPH oxidases in the kidney

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Permselective dysfunction of podocyte-podocyte contact upon angiotensin II unravels the molecular target for renoprotective intervention

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Changes in glomerular perm-selectivity induced by angiotensin II imply podocyte dysfunction and slit diaphragm protein rearrangement

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Stretch reduces nephrin expression via an angiotensin II-AT(1)-dependent mechanism in human podocytes: effect of rosiglitazone

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Mechanical forces and TGFbeta1 reduce podocyte adhesion through alpha3beta1 integrin downregulation.

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Angiotensin II stimulates the synthesis of vascular endothelial growth factor through the p38 mitogen activated protein kinase pathway in cultured mouse podocytes.

21 January 2018

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Pathogenesis of the podocytopathy and proteinuria in diabetic glomerulopathy

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Mechanical stretch induces podocyte hypertrophy in vitro.

21 January 2018

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Angiotensin II receptor blocker inhibits p27Kip1 expression in glucose-stimulated podocytes and in diabetic glomeruli

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Ultrastructural changes in the glomerular filtration barrier and occurrence of proteinuria in Chinese patients with type 2 diabetic nephropathy

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Podocyte biology and response to injury

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Insulin resistance, hyperinsulinemia, and renal injury: mechanisms and implications

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Possible underlying mechanisms responsible for aldosterone and mineralocorticoid receptor-dependent renal injury

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Mineralocorticoid receptor blockade enhances the antiproteinuric effect of an angiotensin II blocker through inhibiting podocyte injury in type 2 diabetic rats.

21 January 2018

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Podocyte as the target for aldosterone: roles of oxidative stress and Sgk1.

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Transcriptional suppression of nephrin in podocytes by macrophages: roles of inflammatory cytokines and involvement of the PI3K/Akt pathway

21 January 2018

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21 January 2018

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Toward the development of podocyte-specific drugs

21 January 2018

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10.3109/01913123.2011.584657

21 January 2018

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21657818%20AND%20SRC:MED&resulttype=core&format=json

3 February 2020

1 reference