Anti-HHV-6 IgG titer significantly predicts subsequent relapse risk in multiple sclerosis (Q37956299)

25 August 2017

title

Anti-HHV-6 IgG titer significantly predicts subsequent relapse risk in multiple sclerosis (English)

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25 August 2017

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Ingrid van der Mei

9

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Leigh Blizzard

5

object named as

Leigh Blizzard

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Anne-Louise Ponsonby

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Terence Dwyer

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Fotini Pittas

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Steve Simpson

1

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Bruce Taylor

2

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Dominic E Dwyer

3

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Janette Taylor

4

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publication date

14 November 2011

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Multiple Sclerosis Journal

25 August 2017

published in

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volume

18

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25 August 2017

issue

6

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25 August 2017

page(s)

799-806

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Relation between Epstein-Barr virus and multiple sclerosis: analytic study of scientific production

25 August 2017

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Human herpesvirus-6 viral load and antibody titer in serum samples of patients with multiple sclerosis.

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Humoral immune response to EBV in multiple sclerosis is associated with disease activity on MRI.

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The immune response against herpesvirus is more prominent in the early stages of MS.

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Association of human herpes virus 6 (HHV-6) with multiple sclerosis: increased IgM response to HHV-6 early antigen and detection of serum HHV-6 DNA.

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Smoking is associated with progressive disease course and increased progression in clinical disability in a prospective cohort of people with multiple sclerosis

21 January 2018

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Differential tropism of human herpesvirus 6 (HHV-6) variants and induction of latency by HHV-6A in oligodendrocytes

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Variant-specific tropism of human herpesvirus 6 in human astrocytes

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Human herpesvirus 6 (HHV-6) induces dysregulation of glutamate uptake and transporter expression in astrocytes

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Mechanism of neuroinflammation: enhanced cytotoxicity and IL-17 production via CD46 binding

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Association of human endogenous retroviruses with multiple sclerosis and possible interactions with herpes viruses

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Endogenous retroviral genes, Herpesviruses and gender in Multiple Sclerosis

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Human endogenous retrovirus (HERV)-W ENV and GAG proteins: physiological expression in human brain and pathophysiological modulation in multiple sclerosis lesions.

21 January 2018

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21 January 2018

Identifiers

DOI

10.1177/1352458511428081

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25 August 2017

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