CD8+ T cells patrol HSV-1-infected trigeminal ganglia and prevent viral reactivation (Q37967563)

26 August 2017

CD8+ T cells patrol HSV-1-infected trigeminal ganglia and prevent viral reactivation (English)

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26 August 2017

Anthony J St Leger

1

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PubMed ID

22161682

26 August 2017

Robert L Hendricks

series ordinal

2

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26 August 2017

8 December 2011

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26 August 2017

7

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inferred from page(s)

published in

Journal of NeuroVirology

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26 August 2017

17

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26 August 2017

528-534

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26 August 2017

6

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https://scigraph.springernature.com/pub.10.1007/s13365-011-0062-1

26 August 2017

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Gamma interferon can block herpes simplex virus type 1 reactivation from latency, even in the presence of late gene expression

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Psychological stress compromises CD8+ T cell control of latent herpes simplex virus type 1 infections

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Two kinetic patterns of epitope-specific CD8 T-cell responses following murine gammaherpesvirus 68 infection

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ICP0 is required for efficient reactivation of herpes simplex virus type 1 from neuronal latency

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Herpes simplex virus turns off the TAP to evade host immunity

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Circulating herpes simplex type 1 (HSV-1)-specific CD8+ T cells do not access HSV-1 latently infected trigeminal ganglia.

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Rates of reactivation of latent herpes simplex virus from mouse trigeminal ganglia ex vivo correlate directly with viral load and inversely with number of infiltrating CD8+ T cells

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Herpes simplex virus type 1 (HSV-1)-induced apoptosis in human dendritic cells as a result of downregulation of cellular FLICE-inhibitory protein and reduced expression of HSV-1 antiapoptotic latency-associated transcript sequences

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Herpes Simplex Virus-Specific Memory CD8+ T Cells Are Selectively Activated and Retained in Latently Infected Sensory Ganglia

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The CD8+ T-cell response to lymphocytic choriomeningitis virus involves the L antigen: uncovering new tricks for an old virus

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Mature dendritic cells infected with herpes simplex virus type 1 exhibit inhibited T-cell stimulatory capacity

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Gamma interferon impedes the establishment of herpes simplex virus type 1 latent infection but has no impact on its maintenance or reactivation in mice.

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Herpes simplex virus epidemiology and ocular importance

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CD8(+) T cells can block herpes simplex virus type 1 (HSV-1) reactivation from latency in sensory neurons

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Immature monocyte-derived dendritic cells are productively infected with herpes simplex virus type 1.

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Cutting Edge: Conventional CD8α+ Dendritic Cells Are Preferentially Involved in CTL Priming After Footpad Infection with Herpes Simplex Virus-1

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Latent herpesvirus infection in human trigeminal ganglia causes chronic immune response

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Herpes simplex virus type 1 latency-associated transcript gene promotes neuronal survival

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Selective retention of herpes simplex virus-specific T cells in latently infected human trigeminal ganglia

21 January 2018

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Memory T cells persisting within the brain after local infection show functional adaptations to their tissue of residence

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A herpes simplex virus type 1 function continuously required for early and late virus RNA synthesis

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Virus-specific CD8+ T cells accumulate near sensory nerve endings in genital skin during subclinical HSV-2 reactivation

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Migratory Dendritic Cells Transfer Antigen to a Lymph Node-Resident Dendritic Cell Population for Efficient CTL Priming

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Polymorphism within the herpes simplex virus (HSV) ribonucleotide reductase large subunit (ICP6) confers type specificity for recognition by HSV type 1-specific cytotoxic T lymphocytes

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12 December 2020

Control of herpes simplex virus type 1 mRNA synthesis in cells infected with wild-type virus or the temperature-sensitive mutant tsK

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Acute viral encephalitis: the recent progress

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Gene expression during reactivation of herpes simplex virus type 1 from latency in the peripheral nervous system is different from that during lytic infection of tissue cultures

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12 December 2020

The quantity of latent viral DNA correlates with the relative rates at which herpes simplex virus types 1 and 2 cause recurrent genital herpes outbreaks.

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12 December 2020

Macrophage control of herpes simplex virus type 1 replication in the peripheral nervous system

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12 December 2020

10.1007/S13365-011-0062-1

Identifiers

DOI

1 reference

26 August 2017

1 reference

PubMed ID

22161682