An environmental analysis of genes associated with schizophrenia: hypoxia and vascular factors as interacting elements in the neurodevelopmental model. (Q37980010)

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scientific article published on 31 January 2012

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English	An environmental analysis of genes associated with schizophrenia: hypoxia and vascular factors as interacting elements in the neurodevelopmental model.	scientific article published on 31 January 2012

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21 January 2020

An environmental analysis of genes associated with schizophrenia: hypoxia and vascular factors as interacting elements in the neurodevelopmental model (English)

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21 January 2020

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21 January 2020

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R Schmidt-Kastner

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21 January 2020

G Esquivel

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21 January 2020

H W M Steinbusch

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31 January 2012

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21 January 2020

Molecular Psychiatry

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21 January 2020

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21 January 2020

1194-1205

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21 January 2020

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Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress.

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HIF-1 and mechanisms of hypoxia sensing

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Hypoxia-regulated differentiation: let's step it up a Notch

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Different networks, common growth factors: shared growth factors and receptors of the vascular and the nervous system

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Gene regulation by hypoxia and the neurodevelopmental origin of schizophrenia

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Systematic meta-analyses and field synopsis of genetic association studies in schizophrenia: the SzGene database

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DNA microarray analysis of cortical gene expression during early recirculation after focal brain ischemia in rat.

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Hypoxia-inducible factor 1 (HIF-1) pathway

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Integration of oxygen signaling at the consensus HRE.

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Brain-specific knock-out of hypoxia-inducible factor-1alpha reduces rather than increases hypoxic-ischemic damage.

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Identification of potential stroke targets by lentiviral vector mediated overexpression of HIF-1 alpha and HIF-2 alpha in a primary neuronal model of hypoxia

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AKT1 Gene Polymorphisms and Obstetric Complications in the Patients with Schizophrenia

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Serious obstetric complications interact with hypoxia-regulated/vascular-expression genes to influence schizophrenia risk.

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Decreased neurotrophic response to birth hypoxia in the etiology of schizophrenia.

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Role of non-neuronal nicotinic acetylcholine receptors in angiogenesis

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Immunohistochemical detection of dysbindin at the astroglial endfeet around the capillaries of mouse brain.

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MLC1 is associated with the dystrophin-glycoprotein complex at astrocytic endfeet

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Genome-wide association study of schizophrenia in a Japanese population

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Ccm1 is required for arterial morphogenesis: implications for the etiology of human cavernous malformations.

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Transcriptional regulation of vascular endothelial cell responses to hypoxia by HIF-1.

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21 January 2018

Oxygen restriction of neonate rats elevates neuregulin-1alpha isoform levels: possible relationship to schizophrenia

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Neuregulin-1, the fetal endothelium, and brain damage in preterm newborns

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Early neurological phenotype in 4 children with biallelic PRODH mutations

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Impaired migration signaling in the hippocampus following prenatal hypoxia.

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Reelin-deficient mice show impaired neurogenesis and increased stroke size

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Identification of a receptor necessary for Nogo-B stimulated chemotaxis and morphogenesis of endothelial cells

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Nogo Receptor 1 (RTN4R) as a candidate gene for schizophrenia: analysis using human and mouse genetic approaches

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Enhanced neurogenesis in the ischemic striatum following EGF-induced expansion of transit-amplifying cells in the subventricular zone.

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DAVID: Database for Annotation, Visualization, and Integrated Discovery

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Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

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A primate-specific, brain isoform of KCNH2 affects cortical physiology, cognition, neuronal repolarization and risk of schizophrenia

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The impact of heat shock protein 70 gene variations on clinical presentation and outcome in schizophrenic inpatients

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Evidence for rare and common genetic risk variants for schizophrenia at protein kinase C, alpha.

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Translocation and down-regulation of protein kinase C-alpha, -beta, and -gamma isoforms during ischemia-reperfusion in rat brain

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Family-based association study of SELENBP1 in schizophrenia

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Comparative gene expression analysis of blood and brain provides concurrent validation of SELENBP1 up-regulation in schizophrenia

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Hypoxia-inducible factor-1alpha suppresses squamous carcinogenic progression and epithelial-mesenchymal transition

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Association of genetic variation in the MET proto-oncogene with schizophrenia and general cognitive ability

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Post-ischemic delayed expression of hepatocyte growth factor and c-Met in mouse brain following focal cerebral ischemia

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Identification of loci associated with schizophrenia by genome-wide association and follow-up

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Strong synaptic transmission impact by copy number variations in schizophrenia

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Rare structural variants in schizophrenia: one disorder, multiple mutations; one mutation, multiple disorders

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Large recurrent microdeletions associated with schizophrenia

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Rare structural variants disrupt multiple genes in neurodevelopmental pathways in schizophrenia

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Rare chromosomal deletions and duplications increase risk of schizophrenia

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Recurrent CNVs disrupt three candidate genes in schizophrenia patients

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Support for the involvement of large copy number variants in the pathogenesis of schizophrenia

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Copy number variation in schizophrenia in the Japanese population

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New copy number variations in schizophrenia.

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Copy number variants in schizophrenia: confirmation of five previous findings and new evidence for 3q29 microdeletions and VIPR2 duplications

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Epigenetics, the epicenter of the hypoxic response

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Role of hypoxia-inducible factors in epigenetic regulation via histone demethylases

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Regulation of Jumonji-domain-containing histone demethylases by hypoxia-inducible factor (HIF)-1alpha

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The histone demethylases JMJD1A and JMJD2B are transcriptional targets of hypoxia-inducible factor HIF.

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Epigenetic mediation of environmental influences in major psychotic disorders

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Hypoxia response and microRNAs: no longer two separate worlds.

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Upregulation of dicer and microRNA expression in the dorsolateral prefrontal cortex Brodmann area 46 in schizophrenia.

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Genome-wide association study identifies five new schizophrenia loci

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Temporal differences in microRNA expression patterns in astrocytes and neurons after ischemic injury

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Fetal hypoxia and structural brain abnormalities in schizophrenic patients, their siblings, and controls.

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Effects of brain-derived neurotrophic factor Val66Met polymorphism on hippocampal volume change in schizophrenia

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Catechol-o-methyl transferase (COMT) val158met polymorphism and adolescent cortical development in patients with childhood-onset schizophrenia, their non-psychotic siblings, and healthy controls.

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DTNBP1 is associated with imaging phenotypes in schizophrenia

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Neuregulin 1 (8p12) and childhood-onset schizophrenia: susceptibility haplotypes for diagnosis and brain developmental trajectories.

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No effect of obstetric complications on basal ganglia volumes in schizophrenia

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Analysis of 94 candidate genes and 12 endophenotypes for schizophrenia from the Consortium on the Genetics of Schizophrenia

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Cytokine and growth factor involvement in schizophrenia--support for the developmental model

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Gene expression and genetic variation in response to endoplasmic reticulum stress in human cells

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A population genetic approach to mapping neurological disorder genes using deep resequencing

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Modelling schizophrenia using human induced pluripotent stem cells

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An exploratory model for G×E interaction on hippocampal volume in schizophrenia; obstetric complications and hypoxia-related genes

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Microarray analysis of acute and delayed gene expression profile in rats after focal ischemic brain injury and reperfusion

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Cerebral cortical thickness and a history of obstetric complications in schizophrenia

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Obstetric complications as a risk factor for first psychotic episodes in childhood and adolescence

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Vascular endothelial cells synthesize and secrete brain-derived neurotrophic factor

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Common variants on chromosome 6p22.1 are associated with schizophrenia

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Common variants conferring risk of schizophrenia

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Association of NPAS3 exonic variation with schizophrenia

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DNA methyltransferase contributes to delayed ischemic brain injury.

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Functional gene group analysis identifies synaptic gene groups as risk factor for schizophrenia.

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Schizophrenia susceptibility alleles are enriched for alleles that affect gene expression in adult human brain

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