Essential roles of gangliosides in the formation and maintenance of membrane microdomains in brain tissues (Q38001328)

26 August 2017

Essential roles of gangliosides in the formation and maintenance of membrane microdomains in brain tissues (English)

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26 August 2017

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Yuhsuke Ohmi

1

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26 August 2017

Yuki Ohkawa

2

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26 August 2017

Yoshio Yamauchi

3

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26 August 2017

Orie Tajima

4

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26 August 2017

Keiko Furukawa

5

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26 August 2017

Koichi Furukawa

6

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26 August 2017

10 April 2012

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26 August 2017

Neurochemical Research

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26 August 2017

37

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26 August 2017

6

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26 August 2017

1185-1191

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https://scigraph.springernature.com/pub.10.1007/s11064-012-0764-7

26 August 2017

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cites work

The role(s) of gangliosides in neural differentiation and repair: a perspective

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

A vital role for glycosphingolipid synthesis during development and differentiation

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Sensory nerve-dominant nerve degeneration and remodeling in the mutant mice lacking complex gangliosides.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Mice lacking complex gangliosides develop Wallerian degeneration and myelination defects

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Reduced motor and sensory functions and emotional response in GM3-only mice: emergence from early stage of life and exacerbation with aging.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Cell-specific deletion of glucosylceramide synthase in brain leads to severe neural defects after birth.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Gangliosides play pivotal roles in the regulation of complement systems and in the maintenance of integrity in nerve tissues

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

The importance of inflammatory mechanisms in Alzheimer disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Neurons express proteins of the classical complement pathway in Alzheimer disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement activation by beta-amyloid in Alzheimer disease

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Functional rafts in cell membranes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement in central nervous system inflammation

21 January 2018

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Complement in Alzheimer's disease: opportunities for modulating protective and pathogenic events

21 January 2018

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The classical complement cascade mediates CNS synapse elimination

21 January 2018

1 reference

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Characterization of C3a and C5a receptors in rat cerebellar granule neurons during maturation. Neuroprotective effect of C5a against apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement: a novel factor in basal and ischemia-induced neurogenesis

21 January 2018

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Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Up-regulated production and activation of the complement system in Alzheimer's disease brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement C5a receptor-mediated signaling may be involved in neurodegeneration in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Myelin-associated glycoprotein and complementary axonal ligands, gangliosides, mediate axon stability in the CNS and PNS: neuropathology and behavioral deficits in single- and double-null mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Gangliosides potentially inhibit extracellular nucleotide metabolism

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Sphingolipids of the nucleus and their role in nuclear signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Effect of ganglioside and tetraspanins in microdomains on interaction of integrins with fibroblast growth factor receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Critical protection from renal ischemia reperfusion injury by CD55 and CD59.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Revitalizing membrane rafts: new tools and insights

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Refractory skin injury in complex knock-out mice expressing only the GM3 ganglioside

21 January 2018

1 reference

12 December 2020

Gangliosides are essential in the protection of inflammation and neurodegeneration via maintenance of lipid rafts: elucidation by a series of ganglioside-deficient mutant mice

1 reference

12 December 2020

b-series Ganglioside deficiency exhibits no definite changes in the neurogenesis and the sensitivity to Fas-mediated apoptosis but impairs regeneration of the lesioned hypoglossal nerve

1 reference

12 December 2020

Respective roles of decay-accelerating factor and CD59 in circumventing glomerular injury in acute nephrotoxic serum nephritis

1 reference

12 December 2020

Induction of decay-accelerating factor by cytokines or the membrane-attack complex protects vascular endothelial cells against complement deposition

1 reference

12 December 2020

GM1-ganglioside-mediated activation of the unfolded protein response causes neuronal death in a neurodegenerative gangliosidosis

1 reference

12 December 2020

Deficiency of complement defense protein CD59 may contribute to neurodegeneration in Alzheimer's disease

1 reference

12 December 2020

10.1007/S11064-012-0764-7

Identifiers

DOI

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Dimensions Publication ID

26 August 2017

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