Activation of the p21(CIP1/WAF1) promoter by bone morphogenetic protein-2 in mouse B lineage cells. (Q38298630)

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9 November 2019

title

Activation of the p21(CIP1/WAF1) promoter by bone morphogenetic protein-2 in mouse B lineage cells (English)

1 reference

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9 November 2019

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5

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9 November 2019

author name string

K Yamato

1

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9 November 2019

S Hashimoto

2

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9 November 2019

T Imamura

3

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9 November 2019

H Uchida

4

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9 November 2019

T Koseki

6

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9 November 2019

A Ishisaki

7

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9 November 2019

M Kizaki

8

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9 November 2019

K Miyazono

9

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9 November 2019

Y Ikeda

10

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9 November 2019

T Nishihara

11

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9 November 2019

language of work or name

English

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publication date

1 July 2001

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9 November 2019

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9 November 2019

volume

20

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9 November 2019

issue

32

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9 November 2019

page(s)

4383-4392

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1204572

9 November 2019

exact match

0 references

cites work

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Bone morphogenetic proteins regulate the developmental program of human hematopoietic stem cells

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Cell growth arrest and induction of cyclin-dependent kinase inhibitor p21 WAF1/CIP1 mediated by STAT1.

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Transforming growth factor beta induces the cyclin-dependent kinase inhibitor p21 through a p53-independent mechanism

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Functional analysis of the transforming growth factor beta responsive elements in the WAF1/Cip1/p21 promoter

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Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene

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WAF1, a potential mediator of p53 tumor suppression

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Bone morphogenetic proteins promote astroglial lineage commitment by mammalian subventricular zone progenitor cells.

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Smad6 is a Smad1/5-induced smad inhibitor. Characterization of bone morphogenetic protein-responsive element in the mouse Smad6 promoter

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Differential inhibition of Smad6 and Smad7 on bone morphogenetic protein- and activin-mediated growth arrest and apoptosis in B cells

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Identification and functional characterization of a Smad binding element (SBE) in the JunB promoter that acts as a transforming growth factor-beta, activin, and bone morphogenetic protein-inducible enhancer

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Characterization of a bone morphogenetic protein-responsive Smad-binding element

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Sp1, but not Sp3, functions to mediate promoter activation by TGF-beta through canonical Sp1 binding sites

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Transcriptional activation of the human p21(WAF1/CIP1) gene by retinoic acid receptor. Correlation with retinoid induction of U937 cell differentiation

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Transcriptional activation of the Cdk inhibitor p21 by vitamin D3 leads to the induced differentiation of the myelomonocytic cell line U937.

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Regulation of the human p21/WAF1/Cip1 promoter in hepatic cells by functional interactions between Sp1 and Smad family members

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Role of Smad proteins and transcription factor Sp1 in p21(Waf1/Cip1) regulation by transforming growth factor-beta.

21 January 2018

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Human papillomavirus type 16 E6 and E7 oncogenes abrogate radiation-induced DNA damage responses in vivo through p53-dependent and p53-independent pathways

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21 January 2018

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inferred from DOI database lookup

7 January 2021

based on heuristic

p53-dependent and independent expression of p21 during cell growth, differentiation, and DNA damage

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204572

inferred from DOI database lookup

7 January 2021

based on heuristic

Dissociation of bone morphogenetic protein-mediated growth arrest and apoptosis of mouse B cells by HPV-16 E6/E7

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204572

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.ONC.1204572

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11466619%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

9 November 2019

0 references

1 reference

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