Differences in gene expression between wild type and Hoxa1 knockout embryonic stem cells after retinoic acid treatment or leukemia inhibitory factor (LIF) removal (Q38330347)

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English	Differences in gene expression between wild type and Hoxa1 knockout embryonic stem cells after retinoic acid treatment or leukemia inhibitory factor (LIF) removal	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

2 September 2017

Differences in gene expression between wild type and Hoxa1 knockout embryonic stem cells after retinoic acid treatment or leukemia inhibitory factor (LIF) removal (English)

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Europe PubMed Central

PubMed publication ID

2 September 2017

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inferred from title

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based on heuristic

inferred from title

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object named as

Pierre Chambon

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author name string

Eduardo Martinez-Ceballos

1

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Europe PubMed Central

PubMed publication ID

2 September 2017

Lorraine J Gudas

3

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Europe PubMed Central

PubMed publication ID

2 September 2017

language of work or name

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publication date

18 February 2005

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Europe PubMed Central

PubMed publication ID

2 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

2 September 2017

280

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Europe PubMed Central

PubMed publication ID

2 September 2017

16

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

16484-16498

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Europe PubMed Central

PubMed publication ID

2 September 2017

The roles of retinoids in vertebrate development

1 reference

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Homeobox genes in normal and malignant cells.

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Colinearity and functional hierarchy among genes of the homeotic complexes

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Chromatin decondensation and nuclear reorganization of the HoxB locus upon induction of transcription

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Sequential activation of HOX2 homeobox genes by retinoic acid in human embryonal carcinoma cells

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Regulation of vertebrate homeobox-containing genes by morphogens.

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An early effect of retinoic acid: cloning of an mRNA (Era-1) exhibiting rapid and protein synthesis-independent induction during teratocarcinoma stem cell differentiation

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Early retinoic acid-induced F9 teratocarcinoma stem cell gene ERA-1: alternate splicing creates transcripts for a homeobox-containing protein and one lacking the homeobox

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Identification of a retinoic acid responsive enhancer 3' of the murine homeobox gene Hox-1.6

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Retinoic acid-responsive enhancers located 3' of the Hox A and Hox B homeobox gene clusters. Functional analysis

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Loss of retinoic acid receptor gamma function in F9 cells by gene disruption results in aberrant Hoxa-1 expression and differentiation upon retinoic acid treatment

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Hox genes in vertebrate development

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Disruption of the Hox-1.6 homeobox gene results in defects in a region corresponding to its rostral domain of expression

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Developmental defects of the ear, cranial nerves and hindbrain resulting from targeted disruption of the mouse homeobox gene Hox-1.6

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Molecular cloning and analysis of a group of genes differentially expressed in cells which overexpress the Hoxa-1 homeobox gene

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An enhancer element in the EphA2 (Eck) gene sufficient for rhombomere-specific expression is activated by HOXA1 and HOXB1 homeobox proteins.

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The recruitment of SOX/OCT complexes and the differential activity of HOXA1 and HOXB1 modulate the Hoxb1 auto-regulatory enhancer function

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An analysis of retinoic acid-induced gene expression and metabolism in AB1 embryonic stem cells

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Homozygous deletion of the CRABPI gene in AB1 embryonic stem cells results in increased CRABPII gene expression and decreased intracellular retinoic acid concentration

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Expression of REX-1, a gene containing zinc finger motifs, is rapidly reduced by retinoic acid in F9 teratocarcinoma cells

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Novel regulators of bone formation: molecular clones and activities

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Mouse GATA-4: a retinoic acid-inducible GATA-binding transcription factor expressed in endodermally derived tissues and heart

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Isolation of cDNA clones specific for collagen IV and laminin from mouse teratocarcinoma cells

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Local alterations of Krox-20 and Hox gene expression in the hindbrain suggest lack of rhombomeres 4 and 5 in homozygote null Hoxa-1 (Hox-1.6) mutant embryos

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Functional dissection of a transcriptionally active, target-specific Hox-Pbx complex

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Pbx proteins display hexapeptide-dependent cooperative DNA binding with a subset of Hox proteins

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Hox gene products modulate the DNA binding activity of Pbx1 and Pbx2

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Pbx-1 Hox heterodimers bind DNA on inseparable half-sites that permit intrinsic DNA binding specificity of the Hox partner at nucleotides 3' to a TAAT motif.

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AbdB-like Hox proteins stabilize DNA binding by the Meis1 homeodomain proteins

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Retinoic acid regulates the expression of PBX1, PBX2, and PBX3 in P19 cells both transcriptionally and post-translationally

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Gene repression by Polycomb group protein complexes: a distinct complex for every occasion?

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Segment-specific expression of the neuronatin gene during early hindbrain development

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Neuronatin mRNA: alternatively spliced forms of a novel brain-specific mammalian developmental gene

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WISP genes are members of the connective tissue growth factor family that are up-regulated in wnt-1-transformed cells and aberrantly expressed in human colon tumors

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Expression of multiple novel Wnt-1/int-1-related genes during fetal and adult mouse development

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The Wnt-1 (int-1) proto-oncogene is required for development of a large region of the mouse brain

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The midbrain-hindbrain phenotype of Wnt-1-/Wnt-1- mice results from stepwise deletion of engrailed-expressing cells by 9.5 days postcoitum

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Wnt-1 inhibits nerve growth factor-induced differentiation of PC12 cells by preventing the induction of some but not all late-response genes

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Differential regulation of midbrain dopaminergic neuron development by Wnt-1, Wnt-3a, and Wnt-5a

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Expression of two members of the Wnt family during mouse development--restricted temporal and spatial patterns in the developing neural tube.

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Interactions of Wnt‐1 and Wnt‐3a are essential for neural tube patterning

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RhoB is expressed in migrating neural crest and endocardial cushions of the developing mouse embryo.

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Molecular pathogenesis of meningiomas.

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Development of trophic interactions in the vertebrate peripheral nervous system

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Developmental gene control of brainstem function: views from the embryo

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Osteoblast-specific factor 2: cloning of a putative bone adhesion protein with homology with the insect protein fasciclin I

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Expression and function of periostin-isoforms in bone

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Identification of a TAAT-containing motif required for high level expression of the COL1A1 promoter in differentiated osteoblasts of transgenic mice

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Use of type I collagen green fluorescent protein transgenes to identify subpopulations of cells at different stages of the osteoblast lineage.

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Characterization of the human bone sialoprotein (BSP) gene and its promoter sequence

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Cloning of a 2.5 kb murine bone sialoprotein promoter fragment and functional analysis of putative Osf2 binding sites

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Murine Hoxc-9 gene contains a structurally and functionally conserved enhancer.

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Neuronal defects in the hindbrain of Hoxa1, Hoxb1 and Hoxb2 mutants reflect regulatory interactions among these Hox genes

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Quantitative expression of Oct-3/4 defines differentiation, dedifferentiation or self-renewal of ES cells

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Myeloid leukaemia inhibitory factor maintains the developmental potential of embryonic stem cells

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Lineage choice and differentiation in mouse embryos and embryonic stem cells

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Retinoic acid inhibits leukemia inhibitory factor signaling pathways in mouse embryonic stem cells

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Differentiation of embryonic stem cells is induced by GATA factors

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Id genes are direct targets of bone morphogenetic protein induction in embryonic stem cells

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Retinoic acid induces stage-specific repatterning of the rostral central nervous system.

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Rhombomere boundaries are Wnt signaling centers that regulate metameric patterning in the zebrafish hindbrain

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Expression of the homeobox Hox-4 genes and the specification of position in chick wing development

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Human HOX genes are differentially activated by retinoic acid in embryonal carcinoma cells according to their position within the four loci

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21 January 2018

Identifiers

10.1074/JBC.M414397200

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

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