Gene expression profiling in chronic copper overload reveals upregulation of Prnp and App. (Q38335773)

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3 January 2020

title

Gene expression profiling in chronic copper overload reveals upregulation of Prnp and App (English)

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3 January 2020

4

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3 January 2020

author name string

Angela D Armendariz

series ordinal

1

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3 January 2020

Mauricio Gonzalez

series ordinal

2

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3 January 2020

Alexander V Loguinov

series ordinal

3

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3 January 2020

publication date

5 October 2004

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3 January 2020

published in

Physiological Genomics

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3 January 2020

volume

20

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3 January 2020

issue

1

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3 January 2020

page(s)

45-54

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Copper depletion down-regulates expression of the Alzheimer's disease amyloid-beta precursor protein gene

3 January 2020

cites work

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Abnormalities of copper accumulation in cell lines established from nine different alleles of mottled are the same as those found in Menkes disease

21 January 2018

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The amyloid precursor protein of Alzheimer's disease in the reduction of copper(II) to copper(I)

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Regulatory mechanisms controlling gene expression mediated by the antioxidant response element

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Microarrays and toxicology: the advent of toxicogenomics

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Metallothionein messenger RNA regulation in the mottled mouse and Menkes kinky hair syndrome

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Endosomal trafficking of the Menkes copper ATPase ATP7A is mediated by vesicles containing the Rab7 and Rab5 GTPase proteins.

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21 January 2018

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Copper in disorders with neurological symptoms: Alzheimer's, Menkes, and Wilson diseases.

21 January 2018

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Research communication copper-1,10-phenanthroline induces internucleosomal DNA fragmentation in HepG2 cells, resulting from direct oxidation by the hydroxyl radical

21 January 2018

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