Diesel exhaust activates redox-sensitive transcription factors and kinases in human airways. (Q38443662)

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9 January 2020

title

Diesel exhaust activates redox-sensitive transcription factors and kinases in human airways (English)

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2

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Anders Blomberg

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author name string

Jamshid Pourazar

1

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James M Samet

3

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Ragnberth Helleday

4

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Susan J Wilson

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Anthony J Frew

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Frank J Kelly

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Thomas Sandström

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publication date

4 March 2005

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American Journal of Physiology - Lung Cellular and Molecular Physiology

9 January 2020

published in

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volume

289

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issue

5

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page(s)

L724-30

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Airborne particles evoke an inflammatory response in human airway epithelium. Activation of transcription factors

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Benzene-extracted components are important for the major activity of diesel exhaust particles: effect on interleukin-8 gene expression in human bronchial epithelial cells

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An in vitro and in vivo investigation of the effects of diesel exhaust on human airway lining fluid antioxidants.

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Diesel exhaust exposure enhances the expression of IL-13 in the bronchial epithelium of healthy subjects.

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Phosphorylation of c-jun mediated by MAP kinases.

21 January 2018

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Size fractions of ambient particulate matter induce granulocyte macrophage colony-stimulating factor in human bronchial epithelial cells by mitogen-activated protein kinase pathways

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Acute Exposure to Diesel Exhaust Increases IL-8 and GRO- α Production in Healthy Human Airways

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Quinoid redox cycling as a mechanism for sustained free radical generation by inhaled airborne particulate matter

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Short-term exposure to air pollution in a road tunnel enhances the asthmatic response to allergen.

21 January 2018

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21 January 2018

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21 January 2018

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Reactive oxygen species and extracellular signal-regulated kinase 1/2 mitogen-activated protein kinase mediate hyperoxia-induced cell death in lung epithelium

21 January 2018

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21 January 2018

Identifiers

DOI

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9 January 2020

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