Transforming growth factor-beta regulation of epithelial tight junction proteins enhances barrier function and blocks enterohemorrhagic Escherichia coli O157:H7-induced increased permeability (Q38702390)

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scientific article published in December 2005

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English	Transforming growth factor-beta regulation of epithelial tight junction proteins enhances barrier function and blocks enterohemorrhagic Escherichia coli O157:H7-induced increased permeability	scientific article published in December 2005

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9 January 2020

Transforming growth factor-beta regulation of epithelial tight junction proteins enhances barrier function and blocks enterohemorrhagic Escherichia coli O157:H7-induced increased permeability (English)

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9 January 2020

Escherichia coli

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9 January 2020

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Kathryn L Howe

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9 January 2020

Arthur Wang

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Aisha Nazli

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Derek M McKay

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1 December 2005

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9 January 2020

The American Journal of Pathology

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9 January 2020

167

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9 January 2020

1587-1597

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9 January 2020

Epithelia under metabolic stress perceive commensal bacteria as a threat

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Claudin extracellular domains determine paracellular charge selectivity and resistance but not tight junction fibril architecture

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Saccharomyces boulardii interferes with enterohemorrhagic Escherichia coli-induced signaling pathways in T84 cells

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Claudin-based tight junctions are crucial for the mammalian epidermal barrier: a lesson from claudin-1-deficient mice

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Enteropathogenic Escherichia coli activates ezrin, which participates in disruption of tight junction barrier function.

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Conversion of zonulae occludentes from tight to leaky strand type by introducing claudin-2 into Madin-Darby canine kidney I cells

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The structure and function of claudins, cell adhesion molecules at tight junctions.

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6 September 2017

Transforming growth factor beta1 ameliorates intestinal epithelial barrier disruption by Cryptosporidium parvum in vitro in the absence of mucosal T lymphocytes

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Exploitation of host cells by enteropathogenic Escherichia coli

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The risk of the hemolytic-uremic syndrome after antibiotic treatment of Escherichia coli O157:H7 infections

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Circulating levels of transforming growth factor-beta1 and lymphokines among children with hemolytic uremic syndrome

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6 September 2017

Direct binding of three tight junction-associated MAGUKs, ZO-1, ZO-2, and ZO-3, with the COOH termini of claudins

1 reference

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6 September 2017

Transforming growth factor-beta1 preserves epithelial barrier function: identification of receptors, biochemical intermediates, and cytokine antagonists

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6 September 2017

Transmembrane proteins in the tight junction barrier.

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6 September 2017

Signal transduction pathways involved in enterohemorrhagic Escherichia coli-induced alterations in T84 epithelial permeability

1 reference

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Regulation of intestinal epithelial barrier function by TGF-beta 1. Evidence for its role in abrogating the effect of a T cell cytokine.

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Enterohemorrhagic Escherichia coli infection induces interleukin-8 production via activation of mitogen-activated protein kinases and the transcription factors NF-kappaB and AP-1 in T84 cells

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Transforming growth factor-beta 1 inhibits non-pathogenic Gram negative bacteria-induced NF-kappa B recruitment to the interleukin-6 gene promoter in intestinal epithelial cells through modulation of histone acetylation

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Enteropathogenic Escherichia coli dephosphorylates and dissociates occludin from intestinal epithelial tight junctions.

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7 January 2021

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10.1016/S0002-9440(10)61243-6

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9 January 2020

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9 January 2020

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9 January 2020

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