Novel regulators of cardiac inflammation: Matricellular proteins expand their repertoire (Q38704770)

5 September 2017

title

Novel regulators of cardiac inflammation: Matricellular proteins expand their repertoire (English)

1 reference

5 September 2017

0 references

Marieke Rienks

1

1 reference

5 September 2017

Anna-Pia Papageorgiou

series ordinal

2

1 reference

5 September 2017

publication date

12 January 2016

1 reference

Journal of Molecular and Cellular Cardiology

5 September 2017

published in

1 reference

5 September 2017

volume

91

1 reference

5 September 2017

page(s)

172-178

1 reference

The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction

5 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

C-type lectin receptors on dendritic cells and Langerhans cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

A novel mechanism of carbohydrate recognition by the C-type lectins DC-SIGN and DC-SIGNR. Subunit organization and binding to multivalent ligands

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Pathogen recognition and inflammatory signaling in innate immune defenses

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Pathogen recognition and innate immunity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Identification of an N-terminal recognition site in TLR9 that contributes to CpG-DNA-mediated receptor activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

"Eat me" and "don't eat me" signals govern the innate immune response and tissue repair in the CNS: emphasis on the critical role of the complement system

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Since there are PAMPs and DAMPs, there must be SAMPs? Glycan “self-associated molecular patterns” dampen innate immunity, but pathogens can mimic them

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Conditional signaling by Toll-like receptor 4.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Endosymbiotic gene transfer: organelle genomes forge eukaryotic chromosomes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The interplay between pathogen-associated and danger-associated molecular patterns: an inflammatory code in cancer?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Viral causes of cardiac inflammation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Viruses as adjuvants for autoimmunity: evidence from Coxsackievirus-induced myocarditis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Human cardiac inflammatory responses triggered by Coxsackie B viruses are mainly Toll-like receptor (TLR) 8-dependent

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Viral myocarditis: from the perspective of the virus

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Stress-activated protein kinases are involved in coxsackievirus B3 viral progeny release

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Inflammation in viral myocarditis: friend or foe?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Stress cytokines: pivotal proteins in immune regulatory networks; Opinion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Molecular identification of a danger signal that alerts the immune system to dying cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Oligosaccharides of Hyaluronan activate dendritic cells via toll-like receptor 4.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Receptor-mediated monitoring of tissue well-being via detection of soluble heparan sulfate by Toll-like receptor 4.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Role of innate and adaptive immune mechanisms in cardiac injury and repair

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

It takes two to tango: monocyte and macrophage duality in the infarcted heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Kinetics of C5a release in cardiac lymph of dogs experiencing coronary artery ischemia-reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Reduction of myocardial infarct size by neutrophil depletion: effect of duration of occlusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Therapeutic siRNA silencing in inflammatory monocytes in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The emerging role of innate immunity in the heart and vascular system: for whom the cell tolls

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Inflammation revisited: inflammation versus resolution of inflammation following myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Monocyte and macrophage heterogeneity in the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Monocytes in heart failure: relationship to a deteriorating immune overreaction or a desperate attempt for tissue repair?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Regulation of macrophage cytokine production by phagocytosis of apoptotic and post-apoptotic cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Immunosuppressive effects of apoptotic cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Enhanced efferocytosis of apoptotic cardiomyocytes through myeloid-epithelial-reproductive tyrosine kinase links acute inflammation resolution to cardiac repair after infarction.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Monocytes: protagonists of infarct inflammation and repair after myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Antigen Processing for MHC Class II Presentation via Autophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Self-antigen presentation by dendritic cells in autoimmunity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Activation of CD4+ T lymphocytes improves wound healing and survival after experimental myocardial infarction in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Differential macrophage polarization in male and female BALB/c mice infected with coxsackievirus B3 defines susceptibility to viral myocarditis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Regulatory T cells protect mice against coxsackievirus-induced myocarditis through the transforming growth factor beta-coxsackie-adenovirus receptor pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Susceptibility to myocarditis is dependent on the response of alphabeta T lymphocytes to coxsackieviral infection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The role of B lymphocytes in coxsackievirus B3 infection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Myocardial infarct-sparing effect of adenosine A2A receptor activation is due to its action on CD4+ T lymphocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Foxp3+ CD4+ T cells improve healing after myocardial infarction by modulating monocyte/macrophage differentiation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Matricellular proteins: an overview

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Matricellular proteins: an overview

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Revisiting the matricellular concept

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The role of tenascin-C in tissue injury and tumorigenesis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The many facets of the matricelluar protein periostin during cardiac development, remodeling, and pathophysiology

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

A thrombospondin-dependent pathway for a protective ER stress response

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Matricellular proteins in cardiac adaptation and disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Structures of thrombospondins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The evolution of thrombospondins and their ligand-binding activities

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Thrombospondin-2 prevents cardiac injury and dysfunction in viral myocarditis through the activation of regulatory T-cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Interactions between CD47 and thrombospondin reduce inflammation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Thrombospondin/CD47 interaction: a pathway to generate regulatory T cells from human CD4+ CD25- T cells in response to inflammation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Thrombospondin cooperates with CD36 and the vitronectin receptor in macrophage recognition of neutrophils undergoing apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

SPARC, a matricellular protein: at the crossroads of cell-matrix communication

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Absence of SPARC results in increased cardiac rupture and dysfunction after acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Pressure overload-induced alterations in fibrillar collagen content and myocardial diastolic function: role of secreted protein acidic and rich in cysteine (SPARC) in post-synthetic procollagen processing

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Recombinant and tissue-derived mouse BM-40 bind to several collagen types and have increased affinities after proteolytic activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Limited cleavage of extracellular matrix protein BM-40 by matrix metalloproteinases increases its affinity for collagens

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Cleavage of the matricellular protein SPARC by matrix metalloproteinase 3 produces polypeptides that influence angiogenesis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Secreted protein acidic and rich in cysteine facilitates age-related cardiac inflammation and macrophage M1 polarization.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Tenascins: regulation and putative functions during pathological stress

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Tenascin-C in human cardiac pathology

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Meet the tenascins: multifunctional and mysterious

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Tenascin supports lymphocyte rolling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Blockade of alpha 5 beta 1 integrins reverses the inhibitory effect of tenascin on chemotaxis of human monocytes and polymorphonuclear leukocytes through three-dimensional gels of extracellular matrix proteins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Tenascin-C is an endogenous activator of Toll-like receptor 4 that is essential for maintaining inflammation in arthritic joint disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Tenascin-C may aggravate left ventricular remodeling and function after myocardial infarction in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Tenascin-C may accelerate cardiac fibrosis by activating macrophages via the integrin αVβ3/nuclear factor-κB/interleukin-6 axis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Tenascin-C aggravates autoimmune myocarditis via dendritic cell activation and Th17 cell differentiation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The modular architecture of a new family of growth regulators related to connective tissue growth factor

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Adhesion-modulating/matricellular ECM protein families: a structural, functional and evolutionary appraisal.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Regulation of proangiogenic factor CCN1 in cardiac muscle: impact of ischemia, pressure overload, and neurohumoral activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

CCN1 protects cardiac myocytes from oxidative stress via beta1 integrin-Akt pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Matricellular signaling molecule CCN1 attenuates experimental autoimmune myocarditis by acting as a novel immune cell migration modulator.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Genomic expression profiling of human inflammatory cardiomyopathy (DCMi) suggests novel therapeutic targets.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Matricellular protein CCN1 activates a proinflammatory genetic program in murine macrophages

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

CCN1: a novel inflammation-regulated biphasic immune cell migration modulator.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The matricellular functions of small leucine-rich proteoglycans (SLRPs).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Isolation of proteoglycans synthesized by rat heart: evidence for the presence of several distinct forms

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The role of biglycan in the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Increased expression of biglycan mRNA in pressure-overloaded rat heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Induction of myocardial biglycan in heart failure in rats--an extracellular matrix component targeted by AT(1) receptor antagonism

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Ontogeny of enhanced decorin levels and distribution within myocardium of failing hearts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Time-dependent changes of decorin in the infarct zone after experimentally induced myocardial infarction in rats: comparison with biglycan

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Time course of collagen and decorin changes in rat cardiac and skeletal muscle post-MI

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Elevation of Expression of Smads 2, 3, and 4, Decorin and TGF-βin the Chronic Phase of Myocardial Infarct Scar Healing

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Biglycan induces the expression of osteogenic factors in human aortic valve interstitial cells via Toll-like receptor-2.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Left ventricular dilation in toll-like receptor 2 deficient mice after myocardial ischemia/reperfusion through defective scar formation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The matrix component biglycan is proinflammatory and signals through Toll-like receptors 4 and 2 in macrophages

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Biglycan, a danger signal that activates the NLRP3 inflammasome via toll-like and P2X receptors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Signaling by the matrix proteoglycan decorin controls inflammation and cancer through PDCD4 and MicroRNA-21

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

A novel role of the lumican core protein in bacterial lipopolysaccharide-induced innate immune response.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Keratocan and lumican regulate neutrophil infiltration and corneal clarity in lipopolysaccharide-induced keratitis by direct interaction with CXCL1.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Microarray gene expression profiles in dilated and hypertrophic cardiomyopathic end-stage heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Human myocardial protein pattern reveals cardiac diseases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Lumican is increased in experimental and clinical heart failure, and its production by cardiac fibroblasts is induced by mechanical and proinflammatory stimuli.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Galectins: structure, function and therapeutic potential

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Galectins: matricellular glycan-binding proteins linking cell adhesion, migration, and survival

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Galectins in innate immunity: dual functions of host soluble beta-galactoside-binding lectins as damage-associated molecular patterns (DAMPs) and as receptors for pathogen-associated molecular patterns (PAMPs).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Differential profile in inflammatory and mineral metabolism biomarkers in patients with ischemic heart disease without classical coronary risk factors.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Galectin-3 and cardiac function in survivors of acute myocardial infarction.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Galectin-1 controls cardiac inflammation and ventricular remodeling during acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Human galectin-3 is a novel chemoattractant for monocytes and macrophages

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Macrophages and galectin 3 play critical roles in CVB3-induced murine acute myocarditis and chronic fibrosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Osteopontin: role in immune regulation and stress responses

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Extracellular matrix remodeling in canine and mouse myocardial infarcts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Differential effects of osteopontin on the cytotoxic activity of macrophages from young and old mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Osteopontin modulates CD44‐dependent chemotaxis of peritoneal macrophages through G‐protein‐coupled receptors: Evidence of a role for an intracellular form of osteopontin

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Penicillium marneffei causes osteopontin-mediated production of interleukin-12 by peripheral blood mononuclear cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Osteopontin promotes fibrosis in dystrophic mouse muscle by modulating immune cell subsets and intramuscular TGF-beta

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Osteopontin: a fibrosis-related marker molecule in cardiac remodeling of enterovirus myocarditis in the susceptible host

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

The neutrophilic leukocyte in wound repair a study with antineutrophil serum

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Wound healing in the PU.1 null mouse--tissue repair is not dependent on inflammatory cells.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Inflammation in cardiac injury, repair and regeneration.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Myocardial extracellular matrix: an ever-changing and diverse entity.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

Danger signals in the initiation of the inflammatory response after myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2016.01.008

10.1016/J.YJMCC.2016.01.008

7 January 2021

Identifiers

DOI

1 reference

5 September 2017

1 reference