β-Arrestin-biased AT1R stimulation promotes extracellular matrix synthesis in renal fibrosis (Q38713058)

5 September 2017

title

β-Arrestin-biased AT1R stimulation promotes extracellular matrix synthesis in renal fibrosis (English)

1 reference

5 September 2017

0 references

Yandao Wang

1

1 reference

5 September 2017

Jieli Huang

2

1 reference

5 September 2017

Xi Liu

3

1 reference

5 September 2017

Yangyang Niu

4

1 reference

5 September 2017

Liqin Zhao

5

1 reference

5 September 2017

Ying Yu

6

1 reference

5 September 2017

Li Zhou

7

1 reference

5 September 2017

Limin Lu

8

1 reference

5 September 2017

Chen Yu

9

1 reference

5 September 2017

publication date

8 March 2017

1 reference

American Journal of Physiology - Renal Physiology

5 September 2017

published in

1 reference

5 September 2017

volume

313

1 reference

5 September 2017

issue

1

1 reference

5 September 2017

page(s)

F1-F8

1 reference

Differential kinetic and spatial patterns of beta-arrestin and G protein-mediated ERK activation by the angiotensin II receptor

5 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

The angiotensin type 1 receptor activates extracellular signal-regulated kinases 1 and 2 by G protein-dependent and -independent pathways in cardiac myocytes and langendorff-perfused hearts.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Differential extracellular signal-regulated kinases 1 and 2 activation by the angiotensin type 1 receptor supports distinct phenotypes of cardiac myocytes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Mechanical stress induces lung fibrosis by epithelial-mesenchymal transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Mechanical Stretch Induces Apoptosis Regulator TRB3 in Cultured Cardiomyocytes and Volume-Overloaded Heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Quantitative phosphoproteomics dissection of seven-transmembrane receptor signaling using full and biased agonists

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Endocytosis of G protein-coupled receptors: roles of G protein-coupled receptor kinases and beta-arrestin proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Beta-arrestins and cell signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

G protein coupled receptors as drug targets: the role of beta-arrestins

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Renin-angiotensin system in the kidney: What is new?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Angiotensin II signal transduction through the AT1 receptor: novel insights into mechanisms and pathophysiology

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Involvement of glomerular renin-angiotensin system (RAS) activation in the development and progression of glomerular injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Role of β-arrestins and arrestin domain-containing proteins in G protein-coupled receptor trafficking

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Crystal structure of rhodopsin bound to arrestin by femtosecond X-ray laser

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Independent beta-arrestin2 and Gq/protein kinase Czeta pathways for ERK stimulated by angiotensin type 1A receptors in vascular smooth muscle cells converge on transactivation of the epidermal growth factor receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

β-arrestin deficiency protects against pulmonary fibrosis in mice and prevents fibroblast invasion of extracellular matrix

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Mitogen activated protein kinases in renal fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Key fibrogenic mediators: old players. Renin–angiotensin system

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Angiotensin II-induced cyclooxygenase 2 expression in rat aorta vascular smooth muscle cells does not require heterotrimeric G protein activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Induction of cardiac fibrosis by β-blocker in G protein-independent and G protein-coupled receptor kinase 5/β-arrestin2-dependent Signaling pathways.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Teaching old receptors new tricks: biasing seven-transmembrane receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Angiotensin II as a morphogenic cytokine stimulating renal fibrogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Depletion of β-arrestin2 in hepatic stellate cells reduces cell proliferation via ERK pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Allosteric modulation of β-arrestin-biased angiotensin II type 1 receptor signaling by membrane stretch

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Independent beta-arrestin 2 and G protein-mediated pathways for angiotensin II activation of extracellular signal-regulated kinases 1 and 2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Functional specialization of beta-arrestin interactions revealed by proteomic analysis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Global phosphorylation analysis of beta-arrestin-mediated signaling downstream of a seven transmembrane receptor (7TMR).

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

Long-term, high-dosage candesartan suppresses inflammation and injury in chronic kidney disease: nonhemodynamic renal protection.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00588.2016

10.1152/AJPRENAL.00588.2016

21 January 2018

Identifiers

DOI

1 reference

5 September 2017

1 reference