Antiglioma effects of N6-isopentenyladenosine, an endogenous isoprenoid end product, through the downregulation of epidermal growth factor receptor. (Q38733038)

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scientific article published on 3 November 2016

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English	Antiglioma effects of N6-isopentenyladenosine, an endogenous isoprenoid end product, through the downregulation of epidermal growth factor receptor.	scientific article published on 3 November 2016

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26 March 2020

Antiglioma effects of N6-isopentenyladenosine, an endogenous isoprenoid end product, through the downregulation of epidermal growth factor receptor (English)

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26 March 2020

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Giuseppe Catapano

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Elena Ciaglia

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Chiara Laezza

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Simona Pisanti

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Mario Vitale

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Vincenzo Seneca

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Giovanni Torelli

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Silvia Franceschelli

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Patrizia Gazzerro

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Maurizio Bifulco

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16 November 2016

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26 March 2020

International Journal of Cancer

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26 March 2020

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A mutant epidermal growth factor receptor common in human glioma confers enhanced tumorigenicity

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Epidermal growth factor receptor signaling intensity determines intracellular protein interactions, ubiquitination, and internalization

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Involvement of Akt/NF-κB pathway in N6-isopentenyladenosine-induced apoptosis in human breast cancer cells.

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N6-isopentenyladenosine arrests tumor cell proliferation by inhibiting farnesyl diphosphate synthase and protein prenylation

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Serine mutations that abrogate ligand-induced ubiquitination and internalization of the EGF receptor do not affect c-Cbl association with the receptor

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N6-isopentenyladenosine affects cytotoxic activity and cytokines production by IL-2 activated NK cells and exerts topical anti-inflammatory activity in mice.

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Degradation of epidermal growth factor receptor mediates dasatinib-induced apoptosis in head and neck squamous cell carcinoma cells

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Identifiers

10.1002/IJC.30505

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26 March 2020

PubMed publication ID

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26 March 2020

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