HO-1 Signaling Activation by Pterostilbene Treatment Attenuates Mitochondrial Oxidative Damage Induced by Cerebral Ischemia Reperfusion Injury. (Q38873745)

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scientific article published on 16 May 2015

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English	HO-1 Signaling Activation by Pterostilbene Treatment Attenuates Mitochondrial Oxidative Damage Induced by Cerebral Ischemia Reperfusion Injury.	scientific article published on 16 May 2015

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scholarly article

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8 March 2020

HO-1 Signaling Activation by Pterostilbene Treatment Attenuates Mitochondrial Oxidative Damage Induced by Cerebral Ischemia Reperfusion Injury (English)

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8 March 2020

reperfusion injury

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inferred from title

cerebral infarction

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based on heuristic

inferred from title

5

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author name string

Yang Yang

1

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Jiayi Wang

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Yue Li

3

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Chongxi Fan

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Lei Zhao

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8 March 2020

Zhenlong Xin

8

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Bodong Wang

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8 March 2020

Guiling Wu

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Xia Li

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Zhiqing Li

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Xu Gao

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Yushu Dong

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Yan Qu

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publication date

16 May 2015

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Molecular Neurobiology

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4

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2339-2353

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8 March 2020

https://scigraph.springernature.com/pub.10.1007/s12035-015-9194-2

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17β-Estradiol prevents cell death and mitochondrial dysfunction by an estrogen receptor-dependent mechanism in astrocytes after oxygen-glucose deprivation/reperfusion

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A review of pterostilbene antioxidant activity and disease modification

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Dietary administration of high doses of pterostilbene and quercetin to mice is not toxic.

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Low-dose pterostilbene, but not resveratrol, is a potent neuromodulator in aging and Alzheimer's disease.

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In planta production of the highly potent resveratrol analogue pterostilbene via stilbene synthase and O-methyltransferase co-expression

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Pterostilbene: Biomedical applications.

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Pterostilbene attenuates lipopolysaccharide-induced learning and memory impairment possibly via inhibiting microglia activation and protecting neuronal injury in mice

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Anxiolytic action of pterostilbene: involvement of hippocampal ERK phosphorylation

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Induction of hemeoxygenase-1 expression after inhibition of hemeoxygenase activity promotes inflammation and worsens ischemic brain damage in mice.

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Up-regulation of heme oxygenase-1 attenuates brain damage after cerebral ischemia via simultaneous inhibition of superoxide production and preservation of NO bioavailability

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Curcumin ameliorates the permeability of the blood-brain barrier during hypoxia by upregulating heme oxygenase-1 expression in brain microvascular endothelial cells.

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Ethyl pyruvate-mediated Nrf2 activation and hemeoxygenase 1 induction in astrocytes confer protective effects via autocrine and paracrine mechanisms

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Resveratrol pretreatment attenuates cerebral ischemic injury by upregulating expression of transcription factor Nrf2 and HO-1 in rats

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Heme oxygenase-1 (HO-1) upregulation delays morphological and oxidative damage induced in an excitotoxic/pro-oxidant model in the rat striatum.

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Induction of heme oxygenase protein protects neurons in cortex and striatum, but not in hippocampus, against transient forebrain ischemia.

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Reduced HO-1 protein expression is associated with more severe neurodegeneration after transient ischemia induced by cortical compression in diabetic Goto-Kakizaki rats.

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21 January 2018

Protective effect of Pterostilbene against free radical mediated oxidative damage

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Pterostilbene protects vascular endothelial cells against oxidized low-density lipoprotein-induced apoptosis in vitro and in vivo

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1 reference

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10.1007/S12035-015-9194-2

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