P53 dependent mitochondrial permeability transition pore opening is required for dexamethasone-induced death of osteoblasts (Q39016683)

8 September 2017

title

P53 dependent mitochondrial permeability transition pore opening is required for dexamethasone-induced death of osteoblasts (English)

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8 September 2017

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Yun-Fang Zhen

1

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8 September 2017

Guo-Dong Wang

2

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Lun-Qing Zhu

3

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Shi-Ping Tan

4

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Fu-Yong Zhang

5

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Xiao-Zhong Zhou

6

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Xiao-Dong Wang

7

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publication date

1 October 2014

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Journal of Cellular Physiology

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language of work

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volume

229

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issue

10

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8 September 2017

page(s)

1475-1483

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Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

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cites work

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Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin-D at a different site from cyclosporin A.

21 January 2018

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Mitochondrial intermembrane junctional complexes and their involvement in cell death

21 January 2018

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Cyclophilin-D binds strongly to complexes of the voltage-dependent anion channel and the adenine nucleotide translocase to form the permeability transition pore

21 January 2018

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Advances in glucocorticoid-induced osteoporosis

21 January 2018

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Calcium, mitochondria and reperfusion injury: a pore way to die.

21 January 2018

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The permeability transition pore complex: another view.

21 January 2018

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Mitochondrial cyclophilin-D as a critical mediator of ischaemic preconditioning

21 January 2018

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Mitochondrial permeability transition and cell death: the role of cyclophilin d

21 January 2018

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Ultra-violet B (UVB)-induced skin cell death occurs through a cyclophilin D intrinsic signaling pathway

21 January 2018

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Glucocorticoids in osteonecrosis of the femoral head: a new understanding of the mechanisms of action.

21 January 2018

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Glucocorticoid receptor and sequential P53 activation by dexamethasone mediates apoptosis and cell cycle arrest of osteoblastic MC3T3-E1 cells

21 January 2018

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Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

21 January 2018

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Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

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Glucocorticoids act directly on osteoblasts and osteocytes to induce their apoptosis and reduce bone formation and strength

21 January 2018

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The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ

21 January 2018

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5,6-Dimethylxanthenone-4-acetic acid (DMXAA) activates stimulator of interferon gene (STING)-dependent innate immune pathways and is regulated by mitochondrial membrane potential

21 January 2018

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Mechanisms involved in the side effects of glucocorticoids

21 January 2018

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Activation of AMPK protects against hydrogen peroxide-induced osteoblast apoptosis through autophagy induction and NADPH maintenance: new implications for osteonecrosis treatment?

21 January 2018

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Cyclosporin A attenuates acute mitochondrial dysfunction following traumatic brain injury.

21 January 2018

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Role of the mitochondrial membrane permeability transition in cell death

21 January 2018

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Identification of a functional link for the p53 tumor suppressor protein in dexamethasone-induced growth suppression

21 January 2018

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p53 opens the mitochondrial permeability transition pore to trigger necrosis

21 January 2018

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Clinical practice. Glucocorticoid-induced bone disease

21 January 2018

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Inhibition of osteoblastogenesis and promotion of apoptosis of osteoblasts and osteocytes by glucocorticoids. Potential mechanisms of their deleterious effects on bone

21 January 2018

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Glucocorticoid induces apoptosis of osteoblast cells through the activation of glycogen synthase kinase 3beta.

21 January 2018

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Is p53 the Long-Sought Molecular Trigger for Cyclophilin D–Regulated Mitochondrial Permeability Transition Pore Formation and Necrosis?

21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

10.1002/JCP.24589

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8 September 2017

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