Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b. (Q39088618)

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scientific article published on 2 October 2013

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English	Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b.	scientific article published on 2 October 2013

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26 February 2020

Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b (English)

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26 February 2020

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author name string

Bo Wang

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Jay C Jha

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Shinji Hagiwara

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Aaron D McClelland

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Karin Jandeleit-Dahm

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Mark E Cooper

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Phillip Kantharidis

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2 October 2013

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Kidney International

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85

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352-361

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https://scigraph.springernature.com/pub.10.1038/ki.2013.372

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Transforming growth factor-beta regulates tubular epithelial-myofibroblast transdifferentiation in vitro

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Diverse roles of TGF-β/Smads in renal fibrosis and inflammation

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Connective tissue growth factor plays an important role in advanced glycation end product-induced tubular epithelial-to-mesenchymal transition: implications for diabetic renal disease.

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Diabetes complications: the microRNA perspective.

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MicroRNA circuits in transforming growth factor-β actions and diabetic nephropathy.

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The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

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MicroRNAs as regulators of epithelial-mesenchymal transition.

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Overexpression of the microRNA hsa-miR-200c leads to reduced expression of transcription factor 8 and increased expression of E-cadherin

1 reference

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The miR-200 family inhibits epithelial-mesenchymal transition and cancer cell migration by direct targeting of E-cadherin transcriptional repressors ZEB1 and ZEB2

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TGF-β/Smad3 signaling promotes renal fibrosis by inhibiting miR-29

1 reference

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Suppression of microRNA-29 expression by TGF-β1 promotes collagen expression and renal fibrosis

1 reference

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7 January 2021

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Loss of MicroRNA-192 promotes fibrogenesis in diabetic nephropathy

1 reference

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miR-192 mediates TGF-beta/Smad3-driven renal fibrosis

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MicroRNA-192 in diabetic kidney glomeruli and its function in TGF-beta-induced collagen expression via inhibition of E-box repressors.

1 reference

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Inhibiting microRNA-192 ameliorates renal fibrosis in diabetic nephropathy

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Identification of microRNA-93 as a novel regulator of vascular endothelial growth factor in hyperglycemic conditions

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miR-21 is a key therapeutic target for renal injury in a mouse model of type 2 diabetes.

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MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

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TGF-beta isoforms in renal fibrogenesis

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miR-200a Prevents renal fibrogenesis through repression of TGF-β2 expression

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SB-431542 is a potent and specific inhibitor of transforming growth factor-beta superfamily type I activin receptor-like kinase (ALK) receptors ALK4, ALK5, and ALK7

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Inhibition of transforming growth factor (TGF)-beta1-induced extracellular matrix with a novel inhibitor of the TGF-beta type I receptor kinase activity: SB-431542

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Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene

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E-cadherin expression is regulated by miR-192/215 by a mechanism that is independent of the profibrotic effects of transforming growth factor-beta

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Role of MicroRNAs in Fibrosis

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MicroRNAs and diabetic complications

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Role of microRNAs in diabetes and its cardiovascular complications.

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Comprehensive gene and microRNA expression profiling reveals a role for microRNAs in human liver development

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The 21-nucleotide let-7 RNA regulates developmental timing in Caenorhabditis elegans

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The let-7 family of microRNAs

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How does Lin28 let-7 control development and disease?

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Functional genomic analysis of the let-7 regulatory network in Caenorhabditis elegans

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An anti-let-7 sponge decoys and decays endogenous let-7 functions

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Induced pluripotent stem cell lines derived from human somatic cells

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FGF regulates TGF-β signaling and endothelial-to-mesenchymal transition via control of let-7 miRNA expression

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Lipoxins attenuate renal fibrosis by inducing let-7c and suppressing TGFβR1.

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Imatinib attenuates diabetic nephropathy in apolipoprotein E-knockout mice

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Comparison of chronic renal failure rats and modification of the preparation protocol as a hyperphosphataemia model

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Identifiers

10.1038/KI.2013.372

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26 February 2020

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26 February 2020

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