Interaction of natural killer cells with Trypanosoma cruzi-infected fibroblasts (Q39179244)

9 September 2017

title

Interaction of natural killer cells with Trypanosoma cruzi-infected fibroblasts (English)

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9 September 2017

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Lieke T

1

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9 September 2017

Steeg C

2

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9 September 2017

Graefe SE

3

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9 September 2017

Fleischer B

4

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9 September 2017

Jacobs T

5

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9 September 2017

publication date

1 August 2006

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Clinical and Experimental Immunology

9 September 2017

published in

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9 September 2017

volume

145

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9 September 2017

issue

2

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9 September 2017

page(s)

357-364

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Chagas disease etiology: autoimmunity or parasite persistence?

9 September 2017

cites work

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Trypanosoma cruzi: Immune destruction of parasitized mouse fibroblasts in vitro

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Host and parasite apoptosis following Trypanosoma cruzi infection in in vitro and in vivo models

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Trypanosoma cruzi does not induce apoptosis in murine fibroblasts

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Immediate/early response to Trypanosoma cruzi infection involves minimal modulation of host cell transcription

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IL-12 and IFN-gamma production, and NK cell activity, in acute and chronic experimental Trypanosoma cruzi infections

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Trypanosoma cruzi is a potent inducer of interleukin-12 production in macrophages

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Trypanosoma cruzi induces strong IL-12 and IL-18 gene expression in vivo: correlation with interferon-gamma (IFN-gamma) production

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NK1.1+ cells and T-cell activation in euthymic and thymectomized C57Bl/6 mice during acute Trypanosoma cruzi infection.

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Pivotal role of interleukin-12 and interferon-gamma axis in controlling tissue parasitism and inflammation in the heart and central nervous system during Trypanosoma cruzi infection

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Interferon-gamma-induced nitric oxide causes intrinsic intestinal denervation in Trypanosoma cruzi-infected mice

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Inducible nitric oxide synthase is not essential for control of Trypanosoma cruzi infection in mice

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Stage-dependent role of nitric oxide in control of Trypanosoma cruzi infection

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Does myoglobin protect Trypanosoma cruzi from the antiparasitic effects of nitric oxide?.

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NK cells contribute to the control of Trypanosoma cruzi infection by killing free parasites by perforin-independent mechanisms

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Fibroblasts as host cells in latent leishmaniosis

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Activation of Toll-like receptor-2 by glycosylphosphatidylinositol anchors from a protozoan parasite

21 January 2018

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Interleukin-12 but not interleukin-18 is required for immunity to Trypanosoma cruzi in mice

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Replication of Toxoplasma gondii, but not Trypanosoma cruzi, is regulated in human fibroblasts activated with gamma interferon: requirement of a functional JAK/STAT pathway

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Defective nitric oxide effector functions lead to extreme susceptibility of Trypanosoma cruzi-infected mice deficient in gamma interferon receptor or inducible nitric oxide synthase

15 September 2018

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Efficient technique for screening drugs for activity against Trypanosoma cruzi using parasites expressing beta-galactosidase

15 September 2018

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Regulation of Trypanosoma cruzi infection in mice by gamma interferon and interleukin 10: role of NK cells

15 September 2018

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Macrophage killing of Leishmania parasite in vivo is mediated by nitric oxide from L-arginine

15 September 2018

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Endogenous IFN-gamma is required for resistance to acute Trypanosoma cruzi infection in mice.

15 September 2018

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Activated macrophages destroy intracellular Leishmania major amastigotes by an L-arginine-dependent killing mechanism

15 September 2018

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PubMed

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inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1111/J.1365-2249.2006.03118.X

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9 September 2017

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