Cellular plasticity confers migratory and invasive advantages to a population of glioblastoma-initiating cells that infiltrate peritumoral tissue. (Q39196178)

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scientific article published on June 2013

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English	Cellular plasticity confers migratory and invasive advantages to a population of glioblastoma-initiating cells that infiltrate peritumoral tissue.	scientific article published on June 2013

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23 February 2020

Cellular plasticity confers migratory and invasive advantages to a population of glioblastoma-initiating cells that infiltrate peritumoral tissue (English)

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23 February 2020

Juan Antonio Montero

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23 February 2020

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23 February 2020

Alberto Elosegui-Artola

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23 February 2020

Santiago Diaz-Moralli

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23 February 2020

Azucena Esparís-Ogando

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23 February 2020

Marta Cascante Serratosa

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23 February 2020

Jose A Martinez-Climent

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23 February 2020

Atanasio Pandiella

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Atanasio Pandiella

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23 February 2020

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Patricia Ruiz-Ontañon

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23 February 2020

Beatriz Aldaz

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23 February 2020

Juan Martino

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23 February 2020

Maria T Berciano

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23 February 2020

Lara Grande

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23 February 2020

Lorena Nogueira

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23 February 2020

Alfonso Vazquez-Barquero

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23 February 2020

Miguel Lafarga

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23 February 2020

Victor Segura

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23 February 2020

Victoria Sanz-Moreno

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23 February 2020

Jose L Fernandez-Luna

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23 February 2020

publication date

1 June 2013

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23 February 2020

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23 February 2020

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23 February 2020

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23 February 2020

1075-1085

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23 February 2020

Cost of Migration: Invasion of Malignant Gliomas and Implications for Treatment

1 reference

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21 January 2018

Imaging-based stereotaxic serial biopsies in untreated intracranial glial neoplasms.

1 reference

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CXCR4 expression is elevated in glioblastoma multiforme and correlates with an increase in intensity and extent of peritumoral T2-weighted magnetic resonance imaging signal abnormalities

1 reference

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Use of in vivo near-infrared laser confocal endomicroscopy with indocyanine green to detect the boundary of infiltrative tumor

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Graphic analysis of microscopic tumor cell infiltration, proliferative potential, and vascular endothelial growth factor expression in an autopsy brain with glioblastoma

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Survival of the fittest: cancer stem cells in therapeutic resistance and angiogenesis

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Cell migration: Rho GTPases lead the way

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In vivo fluorescence resonance energy transfer imaging reveals differential activation of Rho-family GTPases in glioblastoma cell invasion

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Glioblastoma motility occurs in the absence of actin polymer

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Epidermal growth factor-induced enhancement of glioblastoma cell migration in 3D arises from an intrinsic increase in speed but an extrinsic matrix- and proteolysis-dependent increase in persistence

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The mechanical rigidity of the extracellular matrix regulates the structure, motility, and proliferation of glioma cells.

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Integrin signaling to the actin cytoskeleton.

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The protein phosphatase activity of PTEN regulates SRC family kinases and controls glioma migration

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Rigidity sensing at the leading edge through alphavbeta3 integrins and RPTPalpha

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p27Kip1 modulates cell migration through the regulation of RhoA activation.

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Blockade of the NFκB pathway drives differentiating glioblastoma-initiating cells into senescence both in vitro and in vivo.

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Physiologic oxygen concentration enhances the stem-like properties of CD133+ human glioblastoma cells in vitro.

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An impaired transendothelial migration potential of chronic lymphocytic leukemia (CLL) cells can be linked to ephrin-A4 expression

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Rac activation and inactivation control plasticity of tumor cell movement

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Downregulation of both p21/Cip1 and p27/Kip1 produces a more aggressive prostate cancer phenotype

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Differing modes of tumour cell invasion have distinct requirements for Rho/ROCK signalling and extracellular proteolysis

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Involvement of Rac and Rho signaling in cancer cell motility in 3D substrates.

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Affinity of integrins for damaged extracellular matrix: alpha v beta 3 binds to denatured collagen type I through RGD sites

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Cdc42 regulates extracellular matrix remodeling in three dimensions

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p27kip1 controls cell morphology and motility by regulating microtubule-dependent lipid raft recycling

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Rac1 and RhoA promote neurite outgrowth through formation and stabilization of growth cone point contacts.

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