(-)-Epigallocatechin-3-gallate inhibits human papillomavirus (HPV)-16 oncoprotein-induced angiogenesis in non-small cell lung cancer cells by targeting HIF-1α. (Q39215796)

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English	(-)-Epigallocatechin-3-gallate inhibits human papillomavirus (HPV)-16 oncoprotein-induced angiogenesis in non-small cell lung cancer cells by targeting HIF-1α.	scientific article

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scholarly article

1 reference

Europe PubMed Central

10 September 2017

(-)-Epigallocatechin-3-gallate inhibits human papillomavirus (HPV)-16 oncoprotein-induced angiogenesis in non-small cell lung cancer cells by targeting HIF-1α. (English)

1 reference

Europe PubMed Central

10 September 2017

author name string

Li He

1

1 reference

Europe PubMed Central

10 September 2017

Erying Zhang

2

1 reference

Europe PubMed Central

10 September 2017

Jingli Shi

3

1 reference

Europe PubMed Central

10 September 2017

Xiangyong Li

4

1 reference

Europe PubMed Central

10 September 2017

Keyuan Zhou

5

1 reference

Europe PubMed Central

10 September 2017

Qunzhou Zhang

6

1 reference

Europe PubMed Central

10 September 2017

Anh D Le

7

1 reference

Europe PubMed Central

10 September 2017

Xudong Tang

8

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Europe PubMed Central

10 September 2017

publication date

6 January 2013

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Europe PubMed Central

10 September 2017

Cancer Chemotherapy and Pharmacology

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Europe PubMed Central

10 September 2017

71

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Europe PubMed Central

10 September 2017

713-725

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Europe PubMed Central

10 September 2017

3

1 reference

Europe PubMed Central

10 September 2017

https://scigraph.springernature.com/pub.10.1007/s00280-012-2063-z

0 references

Completeness of TNM staging of small-cell and non-small-cell lung cancer in the Danish cancer registry, 2004-2009

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Non-small cell lung cancer in never smokers as a representative 'non-smoking-associated lung cancer': epidemiology and clinical features

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Human papillomavirus type 16 and 18 in primary lung cancers--a meta-analysis

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Human papillomavirus-16 presence and physical status in lung carcinomas from Asia

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Frequent presence of incomplete HPV16 E7 ORFs in lung carcinomas: memories of viral infection.

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Human papillomavirus (HPV) and Merkel cell polyomavirus (MCPyV) in non small cell lung cancer

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Overexpression of human papillomavirus (HPV) type 16 oncoproteins promotes angiogenesis via enhancing HIF-1α and VEGF expression in non-small cell lung cancer cells

1 reference

https://api.crossref.org/works/10.1007%2FS00280-012-2063-Z

21 January 2018

Upregulation of hypoxia-inducible factor-1alpha mRNA and its clinical significance in non-small cell lung cancer.

1 reference

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Expression and clinical implication of HIF-1alpha and VEGF-C in non-small cell lung cancer.

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Targeting heat shock protein 90 overrides the resistance of lung cancer cells by blocking radiation-induced stabilization of hypoxia-inducible factor-1alpha.

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HIF1α regulated expression of XPA contributes to cisplatin resistance in lung cancer

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Association of hypoxia inducible factor-1α polymorphisms with susceptibility to non-small-cell lung cancer.

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HIF, hypoxia and the role of angiogenesis in non-small cell lung cancer

1 reference

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21 January 2018

Epigallocatechin gallate suppresses lung cancer cell growth through Ras-GTPase-activating protein SH3 domain-binding protein 1.

1 reference

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Identification of epigallocatechin-3-gallate in green tea polyphenols as a potent inducer of p53-dependent apoptosis in the human lung cancer cell line A549.

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The green tea polyphenol, epigallocatechin-3-gallate inhibits telomerase and induces apoptosis in drug-resistant lung cancer cells.

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Green tea epigallocatechin-3-gallate inhibits angiogenesis and suppresses vascular endothelial growth factor C/vascular endothelial growth factor receptor 2 expression and signaling in experimental endometriosis in vivo.

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EGCG inhibits the invasion of highly invasive CL1-5 lung cancer cells through suppressing MMP-2 expression via JNK signaling and induces G2/M arrest

1 reference

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21 January 2018

(-)-Epigallocatechin gallate inhibits growth and activation of the VEGF/VEGFR axis in human colorectal cancer cells

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(-)-Epigallocatechin-3-gallate inhibits VEGF expression induced by IL-6 via Stat3 in gastric cancer

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Early transcriptional pattern of angiogenesis induced by EGCG treatment in cervical tumour cells

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Green tea catechins inhibit VEGF-induced angiogenesis in vitro through suppression of VE-cadherin phosphorylation and inactivation of Akt molecule

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Green tea extract and (-)-epigallocatechin-3-gallate inhibit hypoxia- and serum-induced HIF-1alpha protein accumulation and VEGF expression in human cervical carcinoma and hepatoma cells.

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Epigallocatechin gallate inhibits HIF-1alpha degradation in prostate cancer cells

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21 January 2018

Inactivation of prolyl hydroxylase domain (PHD) protein by epigallocatechin (EGCG) stabilizes hypoxia-inducible factor (HIF-1α) and induces hepcidin (Hamp) in rat kidney

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21 January 2018

Green tea polyphenol EGCG suppresses lung cancer cell growth through upregulating miR-210 expression caused by stabilizing HIF-1α.

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Nerve growth factor promotes breast cancer angiogenesis by activating multiple pathways

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Human papillomavirus 16 E5 up-regulates the expression of vascular endothelial growth factor through the activation of epidermal growth factor receptor, MEK/ ERK1,2 and PI3K/Akt

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Targeting VEGF in lung cancer

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Anti-angiogenesis therapy in cancer: current challenges and future perspectives

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Recent agents targeting HIF-1α for cancer therapy

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EGCG inhibits growth of human pancreatic tumors orthotopically implanted in Balb C nude mice through modulation of FKHRL1/FOXO3a and neuropilin

1 reference

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Green tea polyphenol EGCG suppresses cigarette smoke condensate-induced NF-kappaB activation in normal human bronchial epithelial cells.

1 reference

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Overexpression of human papillomavirus type 16 oncoproteins enhances hypoxia-inducible factor 1 alpha protein accumulation and vascular endothelial growth factor expression in human cervical carcinoma cells

1 reference

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Human Papillomavirus Type 16 E7 Up-regulates AKT Activity through the Retinoblastoma Protein

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21 January 2018

Identifiers

10.1007/S00280-012-2063-Z

1 reference

Europe PubMed Central

10 September 2017

1 reference

Europe PubMed Central

10 September 2017

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