Activation of calcium signaling through Trpv1 by nNOS and peroxynitrite as a key trigger of skeletal muscle hypertrophy (Q39234400)

10 September 2017

Activation of calcium signaling through Trpv1 by nNOS and peroxynitrite as a key trigger of skeletal muscle hypertrophy (English)

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10 September 2017

Naoki Ito

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10 September 2017

Urs T Ruegg

2

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10 September 2017

Akira Kudo

3

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10 September 2017

Yuko Miyagoe-Suzuki

4

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10 September 2017

Shin'ichi Takeda

5

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10 September 2017

language of work or name

English

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publication date

2 December 2012

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10 September 2017

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10 September 2017

19

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10 September 2017

101-106

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10 September 2017

1

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https://scigraph.springernature.com/pub.10.1038/nm.3019

10 September 2017

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cites work

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*NO, RSNO, ONOO-, NO+, *NOO, NOx--dynamic regulation of oxidant scavenging, nitric oxide stores, and cyclic GMP-independent cell signaling

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Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

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A phosphatidylinositol 3-kinase/protein kinase B-independent activation of mammalian target of rapamycin signaling is sufficient to induce skeletal muscle hypertrophy

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Primary role of functional ischemia, quantitative evidence for the two-hit mechanism, and phosphodiesterase-5 inhibitor therapy in mouse muscular dystrophy

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Nitric oxide activates TRP channels by cysteine S-nitrosylation.

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S-Glutathiolation by peroxynitrite activates SERCA during arterial relaxation by nitric oxide

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In vivo inhibition of nitric oxide synthase impairs upregulation of contractile protein mRNA in overloaded plantaris muscle

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Ibuprofen inhibits skeletal muscle hypertrophy in rats.

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A key role for NOX4 in epithelial cell death during development of lung fibrosis.

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First in class, potent, and orally bioavailable NADPH oxidase isoform 4 (Nox4) inhibitors for the treatment of idiopathic pulmonary fibrosis

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DHPR alpha1S subunit controls skeletal muscle mass and morphogenesis.

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TRPV1 activation improves exercise endurance and energy metabolism through PGC-1α upregulation in mice

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Vanilloid receptor expressed in the sarcoplasmic reticulum of rat skeletal muscle

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NO production results in suspension-induced muscle atrophy through dislocation of neuronal NOS.

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Amino acids activate mTOR complex 1 via Ca2+/CaM signaling to hVps34.

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mTOR controls mitochondrial oxidative function through a YY1-PGC-1alpha transcriptional complex

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PGC-1alpha protects skeletal muscle from atrophy by suppressing FoxO3 action and atrophy-specific gene transcription

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In-frame dystrophin following exon 51-skipping improves muscle pathology and function in the exon 52-deficient mdx mouse

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NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart.

21 January 2018

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Skeletal muscle NADPH oxidase is increased and triggers stretch-induced damage in the mdx mouse

21 January 2018

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Muscle satellite cells are a functionally heterogeneous population in both somite-derived and branchiomeric muscles.

21 January 2018

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Role of intracellular calcium mobilization and cell-density-dependent signaling in oxidative-stress-induced cytotoxicity in HaCaT keratinocytes.

21 January 2018

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Physiology of nitric oxide in skeletal muscle

21 January 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/23202294

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Nitric oxide-dependence of satellite stem cell activation and quiescence on normal skeletal muscle fibers

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/23202294

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1038/NM.3019

1 reference

10 September 2017

1 reference