Processing of pro-tumor necrosis factor-alpha by venom metalloproteinases: a hypothesis explaining local tissue damage following snake bite (Q39275539)

10 September 2017

title

Processing of pro-tumor necrosis factor-alpha by venom metalloproteinases: a hypothesis explaining local tissue damage following snake bite (English)

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explanatory hypothesis

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7

Theakston RD

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Moura-da-Silva AM

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Laing GD

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Paine MJ

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Dennison JM

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Politi V

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Crampton JM

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language of work or name

English

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publication date

1 September 1996

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European Journal of Immunology

10 September 2017

published in

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volume

26

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issue

9

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10 September 2017

page(s)

2000-2005

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The pathophysiology of tumor necrosis factors

10 September 2017

cites work

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A highly sensitive cell line, WEHI 164 clone 13, for measuring cytotoxic factor/tumor necrosis factor from human monocytes.

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The metzincins — Topological and sequential relations between the astacins, adamalysins, serralysins, and matrixins (collagenases) define a super family of zinc‐peptidases

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Host response to Bothrops asper snake venom. Analysis of edema formation, inflammatory cells, and cytokine release in a mouse model

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Activation of coagulation after administration of tumor necrosis factor to normal subjects

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What turns on the endothelins?

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Tumor necrosis factor-alpha-induced gelatinase B causes delayed opening of the blood-brain barrier: an expanded therapeutic window

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Skeletal Muscle Necrosis and Regeneration after Injection of BaH1, A Hemorrhagic Metalloproteinase Isolated from the Venom of the Snake Bothrops asper (Terciopelo)

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Role of proinflammatory cytokines in a toxic response: application of cytokine knockout mice in toxicological research

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Identifiers

DOI

10.1002/EJI.1830260905

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10 September 2017

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